TY - JOUR
AU - Bacaksiz, Ahmet
AB - Original article 1 1 1 1 Herz 2015 · 40:912–919 Aylin Hatice Yamac · Emrah Sevgili · Sitki Kucukbuzcu · Muharrem Nasifov  · DOI 10.1007/s00059-015-4311-6 1 2 3 1 1 Ziya Ismailoglu · Elif Kilic · Cilem Ercan · Parviz Jafarov · Hüseyin Uyarel · Received: 5 February 2015 Ahmet Bacaksiz Revised: 13 March 2015   Faculty of Medicine, Department of Cardiology, BezmiÂlem Foundation University, Fatih/Istanbul, Turkey Accepted: 22 March 2015   Faculty of Medicine, Department of Biochemistry, BezmiÂlem Foundation University, Istanbul, Turkey Published online: 25 April 2015 © Urban & Vogel 2015   Faculty of Medicine, Department of Medical Biology, BezmiÂlem Foundation University, Istanbul, Turkey Role of cathepsin D activation in major adverse cardiovascular events and new-onset heart failure after STEMI Improvements in the treatment of acute mands in the face of excessive wall stress dex event [20]. Other data revealed that myocardial infarction (AMI), especial- and hypoxia, which can cause a low-ener- ischemic postconditioning induced au- ly the use of reperfusion therapy, have gy state in cardiomyocytes [4]. tophagy, and the activation of autophagy, led to higher numbers of survivors. But Additionally, autophagy may maintain indicated by enhanced levels of activated heart failure after myocardial injury is cardiomyocyte survival after ischemia/hy- CatD, contributed to cardioprotective ef- still a common occurrence despite 
TI - Role of cathepsin D activation in major adverse cardiovascular events and new-onset heart failure after STEMI
JF - Herz
DO - 10.1007/s00059-015-4311-6
DA - 2015-04-25
UR - https://www.deepdyve.com/lp/springer-journals/role-of-cathepsin-d-activation-in-major-adverse-cardiovascular-events-w90H0Dju0L
SP - 912
EP - 920
VL - 40
IS - 6
DP - DeepDyve
ER -