TY - JOUR
AB -  eye in riboflavin and in t,ryptophan deficiency. She notes that despite definite retardation of growth of the whole animal in both deficiencies, measurements of the eyeball indicated that this organ continued at a normal rate of growth. The result was that the deficient animals had relatively large eyes in undersized bodies. That this phenomenon is not grossly apparent is due to recession of the eyeball in the deficient animal. This recession is permitted by the underdevelopment of the Harderian gland. These glands mere approximately half normal size in deficient rats. Sections of the glands from both riboflavin and tryptophan deficient animals showed dense lymphocytic infiltration replacing portions of the glandular tissue. This underdevelopment of the Harderian glands suggested that the corneal vascularization seen in riboflavin and tryptophan deficiency might be due to the lack of Harderian secretion. Accordingly, the glands were removed from the right sockets of 4 healthy rats. The eyes were observed with a biomicroscope for a period of two months subsequently. The cornea remained clear and bright with no signs of drying or vas- cularization. It may be concluded that loss of Harderian secretion is not a major factor in the production of corneal vascularization 
TI - Activity of Pteroylheptaglutamate in Sulfa‐Treated Rats
JF - Nutrition Reviews
DO - 10.1111/j.1753-4887.1949.tb02166.x
DA - 1949-02-01
UR - https://www.deepdyve.com/lp/oxford-university-press/activity-of-pteroylheptaglutamate-in-sulfa-treated-rats-NaUbryYkcV
SP - 61
VL - 7
IS - 2
DP - DeepDyve
ER -