TY - JOUR
AU - Preiss, R.
AB - Intensive Care Med (1998) 24:746-749 © Springer-Verlag 1998 (Narcanti) was given with no significant level. Taking into account the clearly M. Kriiger positive clinical effect, and the patient was shorter half-life of N, the fast formation ki- R. Regenthai admitted to an intensive care unit ICU. netics of NT, and the high level measured M. Richter About 2 h after ingestion, gastric lavage of T, it is obvious that with the increasing R. Preiss was performed with instillation of 30 g ac- duration of poisoning a more unfavourable tivated charcoal and 15 g natrium sutfuri- ratio of the active drugs N/NT in humans cum. Dopamine and norepinephrine had to develops. Because the nature of the antag- be given for 4 h after admission. Forced ar- onism of N at central ~t-opioid receptors is Poisoning and severe tificial respiration (bi-level positive airway competitive, this may explain its ineffec- ventilatory depression pressure, positive end-expiratory pressure tiveness in preventing ventilatory depres- 1 cmH20, respiratory pressure 18 cmH20 sion. We think that this case demonstrates after oral ingestion of the fractional inspired oxygen 0.6, minute ven- that it is very easy to induce dangerous industrially produced analgesic tilation 10.2 1, respiratory rate 14/min) 
TI - Poisoning and severe ventilatory depression after oral ingestion of the industrially produced analgesic mixture tilidine with naloxone (Valoron N solution)
JF - Intensive Care Medicine
DO - 10.1007/s001340050657
DA - 2013-12-27
UR - https://www.deepdyve.com/lp/springer-journals/poisoning-and-severe-ventilatory-depression-after-oral-ingestion-of-INyTobCS0o
SP - 746
EP - 746
VL - 24
IS - 7
DP - DeepDyve
ER -