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Effect of Cortisone on the Influence of Auto-Antibodies on Erythrocyte Enzymes

Effect of Cortisone on the Influence of Auto-Antibodies on Erythrocyte Enzymes Salvidio, E. a n d Rossi, V Acta h a cm at. 22: 286—291 (1959). Institute of Medical Pathology of the University of Genoa (Director: Prof. A. F ie s c h i) E ffect o f C o rtiso n e on th e In flu ence o f A u to -A n ti­ bo dies on E ry th ro c y te E nzym es By E m a n u e l e S a l v i d i o * a n d V i n c e n z o R ossi The real nature of the erythrocyte damage produced by auto­ antibodies in the course of autoimmune haemolytic syndromes has not yet been clarified. It is in fact unknown how the final destruc­ tion of erythrocytes sensitized by anticorpal proteins takes place, though the hypothesis according to which spherocytosis and in­ crease of osmotic fragility can be considered as foresigns, would be justifiable. Such findings might signify the metabolic disturbance of the erythrocyte and contribute to determine the excess of auto­ haemolysis “in vitro” (19), partially correctable by adding glucose or nucleosides to the incubation media (19, 8). It has http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Acta Haematologica Karger

Effect of Cortisone on the Influence of Auto-Antibodies on Erythrocyte Enzymes

Salvidio, Emanuele; Rossi, Vincenzo
Acta Haematologica , Volume 22 (5): 6 – Jan 1, 2009
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Publisher
Karger
Copyright
© 1959 S. Karger AG, Basel
ISSN
0001-5792
eISSN
1421-9662
DOI
10.1159/000205786
Publisher site
See Article on Publisher Site

Abstract

Salvidio, E. a n d Rossi, V Acta h a cm at. 22: 286—291 (1959). Institute of Medical Pathology of the University of Genoa (Director: Prof. A. F ie s c h i) E ffect o f C o rtiso n e on th e In flu ence o f A u to -A n ti­ bo dies on E ry th ro c y te E nzym es By E m a n u e l e S a l v i d i o * a n d V i n c e n z o R ossi The real nature of the erythrocyte damage produced by auto­ antibodies in the course of autoimmune haemolytic syndromes has not yet been clarified. It is in fact unknown how the final destruc­ tion of erythrocytes sensitized by anticorpal proteins takes place, though the hypothesis according to which spherocytosis and in­ crease of osmotic fragility can be considered as foresigns, would be justifiable. Such findings might signify the metabolic disturbance of the erythrocyte and contribute to determine the excess of auto­ haemolysis “in vitro” (19), partially correctable by adding glucose or nucleosides to the incubation media (19, 8). It has

Journal

Acta Haematologica – Karger

Published: Jan 1, 2009

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