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Metabolic effects of sucralose

Metabolic effects of sucralose Resea R ch h ighlights D ia B ete S n U trit i O n Restoring β-cells Metabolic effects of Dedifferentiation of β­cells is thought to be sucralose a major mechanism underlying β­cell The effects of consuming low-calorie with carbohydrate, but not without loss and dysfunction in sweeteners such as sucralose are the carbohydrate, resulted in reduced type 1 and type 2 diabe­ widely debated, with the literature insulin sensitivity. The participants tes mellitus; however, containing conflicting studies. A new also underwent fMRI, which showed whether this process paper suggests that when sucralose that the participants who consumed could be targeted phar­ is consumed with carbohydrates, sucralose with maltodextrin had The image shows a pancreatic islet from a ma cologically was insulin sensitivity is impaired in reduced responses to sweet taste mouse with streptozotocin-induced diabetes unknown. New research mellitus, with staining for the most frequent cell healthy humans. (but not sour, salty or savoury types in these islets. White stains for the cell nucleus demonstrates that β­cell The researchers randomly tastes) in the midbrain, insular (DAPI staining). β-cells (insulin) are stained green, dedifferentiation can assigned 45 healthy volunteers to and cingulate areas of the brain. α-cells (glucagon) are red http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Nature Reviews Endocrinology Springer Journals

Metabolic effects of sucralose

Nature Reviews Endocrinology , Volume 16 (5) – May 16, 2020

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References (1)

Publisher
Springer Journals
Copyright
Copyright © Springer Nature Limited 2020
ISSN
1759-5029
eISSN
1759-5037
DOI
10.1038/s41574-020-0348-6
Publisher site
See Article on Publisher Site

Abstract

Resea R ch h ighlights D ia B ete S n U trit i O n Restoring β-cells Metabolic effects of Dedifferentiation of β­cells is thought to be sucralose a major mechanism underlying β­cell The effects of consuming low-calorie with carbohydrate, but not without loss and dysfunction in sweeteners such as sucralose are the carbohydrate, resulted in reduced type 1 and type 2 diabe­ widely debated, with the literature insulin sensitivity. The participants tes mellitus; however, containing conflicting studies. A new also underwent fMRI, which showed whether this process paper suggests that when sucralose that the participants who consumed could be targeted phar­ is consumed with carbohydrates, sucralose with maltodextrin had The image shows a pancreatic islet from a ma cologically was insulin sensitivity is impaired in reduced responses to sweet taste mouse with streptozotocin-induced diabetes unknown. New research mellitus, with staining for the most frequent cell healthy humans. (but not sour, salty or savoury types in these islets. White stains for the cell nucleus demonstrates that β­cell The researchers randomly tastes) in the midbrain, insular (DAPI staining). β-cells (insulin) are stained green, dedifferentiation can assigned 45 healthy volunteers to and cingulate areas of the brain. α-cells (glucagon) are red

Journal

Nature Reviews EndocrinologySpringer Journals

Published: May 16, 2020

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