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Severe digital necrosis as the clinical onset of antiphospholipid syndrome

Severe digital necrosis as the clinical onset of antiphospholipid syndrome Intern Emerg Med (2016) 11:761–762 DOI 10.1007/s11739-015-1335-0 CE - M EDI C AL ILLUSTRATION Severe digital necrosis as the clinical onset of antiphospholipid syndrome 1 1 1 • • • Jose ´ Antonio Vargas-Hitos Jose ´ Mario Sabio Nuria Navarrete-Navarrete 1 1 Mo ´ nica Zamora-Pasadas Juan Jime ´nez-Alonso Received: 23 September 2015 / Accepted: 5 October 2015 / Published online: 22 October 2015 SIMI 2015 A 70-year-old man, non-smoker, presented with bilateral improvements in the pain and reperfusion of the base of the digital necrosis, which had developed over the prior month, fingers were observed (Fig. 1b). The APS is a prothrom- related to an aggressive Raynaud’s phenomenon of botic disorder characterized by the predisposition to venous 2 months of evolution (Fig. 1a). The patient had no familial or personal history of previous thrombosis events, heart valve disease, livedo reticularis, nephropathy, thrombocytopenia or neurological manifestations. Upon examination, pulses were present, capillaroscopy was normal, and there was no sclerodactyly or skin sclerosis. Treatment with nifedipine, endovenous alprostidil, bosen- tan and analgesics was initiated with poor results. A complete blood count and biochemical tests, urinalysis, immunoglobulin, serum protein electrophoresis, cryoglob- ulin, serology for hepatitis B and C, antinuclear and antineutrophil http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Internal and Emergency Medicine Springer Journals

Severe digital necrosis as the clinical onset of antiphospholipid syndrome

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References (3)

Publisher
Springer Journals
Copyright
Copyright © 2015 by SIMI
Subject
Medicine & Public Health; Internal Medicine
ISSN
1828-0447
eISSN
1970-9366
DOI
10.1007/s11739-015-1335-0
pmid
26494470
Publisher site
See Article on Publisher Site

Abstract

Intern Emerg Med (2016) 11:761–762 DOI 10.1007/s11739-015-1335-0 CE - M EDI C AL ILLUSTRATION Severe digital necrosis as the clinical onset of antiphospholipid syndrome 1 1 1 • • • Jose ´ Antonio Vargas-Hitos Jose ´ Mario Sabio Nuria Navarrete-Navarrete 1 1 Mo ´ nica Zamora-Pasadas Juan Jime ´nez-Alonso Received: 23 September 2015 / Accepted: 5 October 2015 / Published online: 22 October 2015 SIMI 2015 A 70-year-old man, non-smoker, presented with bilateral improvements in the pain and reperfusion of the base of the digital necrosis, which had developed over the prior month, fingers were observed (Fig. 1b). The APS is a prothrom- related to an aggressive Raynaud’s phenomenon of botic disorder characterized by the predisposition to venous 2 months of evolution (Fig. 1a). The patient had no familial or personal history of previous thrombosis events, heart valve disease, livedo reticularis, nephropathy, thrombocytopenia or neurological manifestations. Upon examination, pulses were present, capillaroscopy was normal, and there was no sclerodactyly or skin sclerosis. Treatment with nifedipine, endovenous alprostidil, bosen- tan and analgesics was initiated with poor results. A complete blood count and biochemical tests, urinalysis, immunoglobulin, serum protein electrophoresis, cryoglob- ulin, serology for hepatitis B and C, antinuclear and antineutrophil

Journal

Internal and Emergency Medicine – Springer Journals

Published: Oct 22, 2015

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