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Science Has Not Proven That Screen Use Impacts Children's Brain Development—Reply

Science Has Not Proven That Screen Use Impacts Children's Brain Development—Reply Letters can alter the expected trajectory of a child’s academic Electronically generated EMFs are often generated in a co- achievements. Importantly, they have implicated polymor- herent state at a specific frequency, in a specific direction, with phisms in the gene network regulating the dopamine trans- a specific phase and specific polarity. Each of these proper- porter protein. This finding requires replication in larger co- ties can be important in EMFs, producing much higher elec- horts, and in cohorts of neonates with HIE. Previous attempts trical forces on electrically charged groups in our bodies, as to find underlying genetic mutations in HIE have been compared with incoherent natural EMFs. Such low-intensity unsuccessful. Focusing on known expression-based poly- EMFs have been shown to produce effects at the cellular level genic risk scores for genes, such as DAT1, offers the potential by activation voltage-gated calcium channels and other volt- to identify mechanisms by which early hypoxic exposure can age-gated ion channels, as well as plant 2-pore channels. Each derail the genetic potential of a child. Understanding these of these channels contain a similar voltage sensor that is mechanisms may help us to identify a broader group of in- thought to be the http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png JAMA Pediatrics American Medical Association

Science Has Not Proven That Screen Use Impacts Children's Brain Development—Reply

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References (5)

Publisher
American Medical Association
Copyright
Copyright 2020 American Medical Association. All Rights Reserved.
ISSN
2168-6203
eISSN
2168-6211
DOI
10.1001/jamapediatrics.2020.0628
Publisher site
See Article on Publisher Site

Abstract

Letters can alter the expected trajectory of a child’s academic Electronically generated EMFs are often generated in a co- achievements. Importantly, they have implicated polymor- herent state at a specific frequency, in a specific direction, with phisms in the gene network regulating the dopamine trans- a specific phase and specific polarity. Each of these proper- porter protein. This finding requires replication in larger co- ties can be important in EMFs, producing much higher elec- horts, and in cohorts of neonates with HIE. Previous attempts trical forces on electrically charged groups in our bodies, as to find underlying genetic mutations in HIE have been compared with incoherent natural EMFs. Such low-intensity unsuccessful. Focusing on known expression-based poly- EMFs have been shown to produce effects at the cellular level genic risk scores for genes, such as DAT1, offers the potential by activation voltage-gated calcium channels and other volt- to identify mechanisms by which early hypoxic exposure can age-gated ion channels, as well as plant 2-pore channels. Each derail the genetic potential of a child. Understanding these of these channels contain a similar voltage sensor that is mechanisms may help us to identify a broader group of in- thought to be the

Journal

JAMA PediatricsAmerican Medical Association

Published: Aug 11, 2020

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