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Pathophysiologic Mechanisms of Cerebral Ischemia and Diffusion Hypoxia in Traumatic Brain Injury.JAMA neurology, 73 5
- Publisher
- American Medical Association
- Copyright
- Copyright 2016 American Medical Association. All Rights Reserved. Applicable FARS/DFARS Restrictions Apply to Government Use.
- ISSN
- 2168-6149
- eISSN
- 2168-6157
- DOI
- 10.1001/jamaneurol.2016.3263
- pmid
- 27618635
- Publisher site
- See Article on Publisher Site
Abstract
Letters Table. Summary of Patient Data Disabling Epilepsy Life Seizures/ Severity Satisfaction Patient mo, No. (Worst, 10) (Best, 10) Age, y/ Seizure Follow-up, Sex Lesion Type mo Before After Before After Before After Comments 56/M Scattered Reflex 3.3 NA NA 8 2 4 9 Unable to walk with encephalomalacia right foot for >35 y; now walks with right foot 9/M Left parietal FCD FDS 7.6 6 0 9 7 5 5 NA 14/M Right parietal FCD Focal motor 7.9 4 1 6 1 2 8 NA 15/F Left MCA infarct FDS 6.7 1 0.5 4 7 7 6 Seizure frequency improved but seizures more severe 26/M Left frontal FCD FDS 6.7 1.5 1 7 3 7 8 NA 27/F Right temporal FCD FDS 9.0 8 0 7 0 4 7 NA 39/F Left parietal EPC motor 7.3 NA NA 10 4 7 6 Seizures now not atrophy noticeable to others; reduced satisfaction from chemotherapy 6/F Right frontal FCD FDS 8.4 8 0 8 0 4 9 NA 14/M Left central FCD FDS 10.7 12 2 4 2 6 8 NA 27/M Right mesial Reflex 16.2 NA NA 8 0 2 4 Unable to walk for parietal FCD >15 y;
Journal
JAMA Neurology – American Medical Association
Published: Nov 12, 2016