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Autosomal Recessive Cerebellar Ataxia 3 Due to Homozygote c.132dupA Mutation Within the ANO10 Gene

Autosomal Recessive Cerebellar Ataxia 3 Due to Homozygote c.132dupA Mutation Within the ANO10 Gene Letters COMMENT & RESPONSE rate ratios were 0.84 (95% CI, 0.42-1.50; P = .65), 0.98 (95% CI, 0.86-1.11; P = .82), and 0.88 (95% CI, 0.77-1.00; P = .06) for al- Multiple Sclerosis and Alcohol Misuse cohol use, abuse, and dependence, respectively. Although not To the Editor I read with interest the article by Pakpoor and significant, low rates of alcohol misuse disorders in the later colleagues investigating the risk for hospital admission for years after first MS diagnosis lead us to speculate that per- multiple sclerosis (MS) after admission for alcohol misuse. haps, after a diagnosis of MS, people with it become more While interesting, the study is far from proving a causal rela- health conscious. These findings—combined with our previ- tionship of alcohol misuse on MS risk. As likely seen in an- ous results that there was a significantly elevated risk for MS other study by the same team, there is an issue of reverse cau- within 1 year of first admission for alcohol abuse only and a sality that is not remedied by excluding admissions in the same significantly elevated risk for MS following all alcohol misuse year because MS risk factors likely act many years http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png JAMA Neurology American Medical Association

Autosomal Recessive Cerebellar Ataxia 3 Due to Homozygote c.132dupA Mutation Within the ANO10 Gene

JAMA Neurology , Volume 72 (2) – Feb 1, 2015

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References (3)

Publisher
American Medical Association
Copyright
Copyright 2015 American Medical Association. All Rights Reserved. Applicable FARS/DFARS Restrictions Apply to Government Use.
ISSN
2168-6149
eISSN
2168-6157
DOI
10.1001/jamaneurol.2014.3918
pmid
25664549
Publisher site
See Article on Publisher Site

Abstract

Letters COMMENT & RESPONSE rate ratios were 0.84 (95% CI, 0.42-1.50; P = .65), 0.98 (95% CI, 0.86-1.11; P = .82), and 0.88 (95% CI, 0.77-1.00; P = .06) for al- Multiple Sclerosis and Alcohol Misuse cohol use, abuse, and dependence, respectively. Although not To the Editor I read with interest the article by Pakpoor and significant, low rates of alcohol misuse disorders in the later colleagues investigating the risk for hospital admission for years after first MS diagnosis lead us to speculate that per- multiple sclerosis (MS) after admission for alcohol misuse. haps, after a diagnosis of MS, people with it become more While interesting, the study is far from proving a causal rela- health conscious. These findings—combined with our previ- tionship of alcohol misuse on MS risk. As likely seen in an- ous results that there was a significantly elevated risk for MS other study by the same team, there is an issue of reverse cau- within 1 year of first admission for alcohol abuse only and a sality that is not remedied by excluding admissions in the same significantly elevated risk for MS following all alcohol misuse year because MS risk factors likely act many years

Journal

JAMA NeurologyAmerican Medical Association

Published: Feb 1, 2015

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