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Another Report of Familial Spontaneous Coronary Artery Dissection

Another Report of Familial Spontaneous Coronary Artery Dissection Letters 2,3 Corresponding Author: Kirsti Uusi-Rasi, PhD, The UKK Institute for Health identified. However, the etiology of SCAD remains Promotion Research, PO Box 30, FI-33501 Tampere, Finland unknown. We feel that genetic study of these 6 families rep- (kirsti.uusi-rasi@uta.fi). resents an exciting opportunity to potentially illuminate the Conflict of Interest Disclosures: None reported. molecular basis of this enigmatic condition of SCAD. Only 2 1. Glendenning P, Zhu K, Inderjeeth C, Howat P, Lewis JR, Prince RL. Effects of individuals have SCAD in any of the 6 pedigrees described, so three-monthly oral 150,000 IU cholecalciferol supplementation on falls, traditional genetic methods would not be successful in iden- mobility, and muscle strength in older postmenopausal women: a randomized controlled trial. J Bone Miner Res. 2012;27(1):170-176. tifying causal genetic variations. However, next-generation 2. Stockton KA, Mengersen K, Paratz JD, Kandiah D, Bennell KL. Effect of sequencing (eg, exome sequencing in combination with bio- vitamin D supplementation on muscle strength: a systematic review and informatic analysis) does have the potential to identify meta-analysis. Osteoporos Int. 2011;22(3):859-871. genetic variations that may contribute to the mechanism of 3. Theodoratou E, Tzoulaki I, Zgaga L, Ioannidis JP. Vitamin D and multiple SCAD. Any candidate genes identified http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png JAMA Internal Medicine American Medical Association

Another Report of Familial Spontaneous Coronary Artery Dissection

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References (14)

Publisher
American Medical Association
Copyright
Copyright 2015 American Medical Association. All Rights Reserved. Applicable FARS/DFARS Restrictions Apply to Government Use.
ISSN
2168-6106
eISSN
2168-6114
DOI
10.1001/jamainternmed.2015.3824
pmid
26436743
Publisher site
See Article on Publisher Site

Abstract

Letters 2,3 Corresponding Author: Kirsti Uusi-Rasi, PhD, The UKK Institute for Health identified. However, the etiology of SCAD remains Promotion Research, PO Box 30, FI-33501 Tampere, Finland unknown. We feel that genetic study of these 6 families rep- (kirsti.uusi-rasi@uta.fi). resents an exciting opportunity to potentially illuminate the Conflict of Interest Disclosures: None reported. molecular basis of this enigmatic condition of SCAD. Only 2 1. Glendenning P, Zhu K, Inderjeeth C, Howat P, Lewis JR, Prince RL. Effects of individuals have SCAD in any of the 6 pedigrees described, so three-monthly oral 150,000 IU cholecalciferol supplementation on falls, traditional genetic methods would not be successful in iden- mobility, and muscle strength in older postmenopausal women: a randomized controlled trial. J Bone Miner Res. 2012;27(1):170-176. tifying causal genetic variations. However, next-generation 2. Stockton KA, Mengersen K, Paratz JD, Kandiah D, Bennell KL. Effect of sequencing (eg, exome sequencing in combination with bio- vitamin D supplementation on muscle strength: a systematic review and informatic analysis) does have the potential to identify meta-analysis. Osteoporos Int. 2011;22(3):859-871. genetic variations that may contribute to the mechanism of 3. Theodoratou E, Tzoulaki I, Zgaga L, Ioannidis JP. Vitamin D and multiple SCAD. Any candidate genes identified

Journal

JAMA Internal Medicine – American Medical Association

Published: Oct 1, 2015

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