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Glomerulomegaly in a Patient With Cyanotic Congenital Heart Disease

Glomerulomegaly in a Patient With Cyanotic Congenital Heart Disease Abstract A 17-year-old boy developed proteinuria nine years after he underwent a Potts-Smith-Gibson procedure as treatment for hypoplastic right ventricle, pulmonary stenosis, and tricuspid atresia. The only abnormality found to account for the proteinuria was glomerulomegaly, a benign lesion previously undescribed in living subjects. References 1. Bauer WC, Rosenberg F: A quantitative study of glomerular enlargement without an increase in renal mass . Amer J Path 37:695-712, 1960. 2. Marinozzi V: Aspetti morfologici del rene nelle cardiopatie congenite cianotizzanti . Policlinico (Med) 66:48-62, 1959. 3. Meessen H, Litton MD: Morphology of the kidney in morbus caeruleus . Arch Path 56:480-487, 1953. 4. Spear GS: The glomerulus in cyanotic congenital heart disease and primary pulmonary hypertension: A review . Nephron 1:238-248, 1964.Crossref 5. Spear GS: Glomerular alterations in cyanotic congenital heart disease . Bull Hopkins Hosp 106: 347-367, 1960. 6. Drummond KN, Vernier RL, Worthen HG, et al: The associated occurrence of the nephrotic syndrome and congenital heart disease . Pediatrics 31:103-114, 1963. 7. Scott HW Jr, Elliott SR II: Renal hemodynamics in congenital cyanotic heart disease . Bull Hopkins Hosp 86:58-71, 1950. http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png American Journal of Diseases of Children American Medical Association

Glomerulomegaly in a Patient With Cyanotic Congenital Heart Disease

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References (9)

Publisher
American Medical Association
Copyright
Copyright © 1970 American Medical Association. All Rights Reserved.
ISSN
0002-922X
DOI
10.1001/archpedi.1970.02100060103017
Publisher site
See Article on Publisher Site

Abstract

Abstract A 17-year-old boy developed proteinuria nine years after he underwent a Potts-Smith-Gibson procedure as treatment for hypoplastic right ventricle, pulmonary stenosis, and tricuspid atresia. The only abnormality found to account for the proteinuria was glomerulomegaly, a benign lesion previously undescribed in living subjects. References 1. Bauer WC, Rosenberg F: A quantitative study of glomerular enlargement without an increase in renal mass . Amer J Path 37:695-712, 1960. 2. Marinozzi V: Aspetti morfologici del rene nelle cardiopatie congenite cianotizzanti . Policlinico (Med) 66:48-62, 1959. 3. Meessen H, Litton MD: Morphology of the kidney in morbus caeruleus . Arch Path 56:480-487, 1953. 4. Spear GS: The glomerulus in cyanotic congenital heart disease and primary pulmonary hypertension: A review . Nephron 1:238-248, 1964.Crossref 5. Spear GS: Glomerular alterations in cyanotic congenital heart disease . Bull Hopkins Hosp 106: 347-367, 1960. 6. Drummond KN, Vernier RL, Worthen HG, et al: The associated occurrence of the nephrotic syndrome and congenital heart disease . Pediatrics 31:103-114, 1963. 7. Scott HW Jr, Elliott SR II: Renal hemodynamics in congenital cyanotic heart disease . Bull Hopkins Hosp 86:58-71, 1950.

Journal

American Journal of Diseases of ChildrenAmerican Medical Association

Published: Jul 1, 1970

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