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H. Blumgart, A. Ernstene (1932)
HEMANGIECTATIC HYPERTROPHY AND CONGENITAL PHLEBARTERIECTASIS: WITH PARTICULAR REFERENCE TO THE DIAGNOSTIC IMPORTANCE OF THE PERIPHERAL VASCULAR PHENOMENAJAMA Internal Medicine, 49
Paul Mason, E. Allen (1935)
CONGENITAL LYMPHANGIECTASIS (LYMPHEDEMA)JAMA Pediatrics, 50
H. Neuhof, M. Berck (1935)
STAPHYLOCOCCIC EMPYEMA AND PYOPNEUMOTHORAX: PATHOGENESIS, PATHOLOGY, SYMPTOMS AND TREATMENTArchives of Surgery, 30
E. Wakefield, E. Hines (1933)
CONGENITAL HEMIHYPERTROPHY: A REPORT OF EIGHT CASESThe American Journal of the Medical Sciences, 185
C. Dent (1911)
“Congenital Elephantiasis” of the ArmJournal of the Royal Society of Medicine, 4
W. Porter, J. Baker (1937)
AN ARTICLE CONTRIBUTED TO AN ANNIVERSARY VOLUME IN HONOR OF DOCTOR JOSEPH HERSEY PRATT: THE SIGNIFICANCE OF CARDIAC ENLARGEMENT CAUSED BY ARTERIOVENOUS FISTULAAnnals of Internal Medicine, 11
B. Horton (1932)
HEMIHYPERTROPHY OF EXTREMITIES ASSOCIATED WITH CONGENITAL ARTERIOVENOUS FISTULAJAMA, 98
H. Faber, Harry Lusignan (1933)
HEREDITARY ELEPHANTIASIS: CONGENITAL ELEPHANTIASIS, HEREDITARY EDEMA, TROPHEDEMA, MILROY'S DISEASEJAMA Pediatrics, 46
H. Ruh, L. Dembo (1925)
CONGENITAL LYMPHANGIECTATIC EDEMAJAMA, 84
George Brown (1929)
ABNORMAL ARTERIOVENOUS COMMUNICATIONS: DIAGNOSED FROM THE OXYGEN CONTENT OF THE BLOOD OF THE REGIONAL VEINSArchives of Surgery, 18
E. Allen (1934)
LYMPHEDEMA OF THE EXTREMITIES: CLASSIFICATION, ETIOLOGY AND DIFFERENTIAL DIAGNOSIS: A STUDY OF THREE HUNDRED CASESJAMA Internal Medicine, 54
J. Veal, W. Mccord (1936)
CONGENITAL ABNORMAL ARTERIOVENOUS ANASTOMOSES OF THE EXTREMITIES: WITH SPECIAL REFERENCE TO DIAGNOSIS BY ARTERIOGRAPHY AND BY THE OXYGEN SATURATION TESTArchives of Surgery, 33
D. Lewis (1930)
CONGENITAL ARTERIOVENOUS FISTULÆ.The Lancet, 216
M. Reid (1925)
STUDIES ON ABNORMAL ARTERIOVENOUS COMMUNICATIONS, ACQUIRED AND CONGENITAL: I. REPORT OF A SERIES OF CASESArchives of Surgery, 10
In the past, there has been a tendency to designate as elephantiasis all types of overgrowth of the extremities without proper regard to the existence of various kinds of hypertrophy, distinct from the point of view of their differing etiologic associations. That there is a real need of applying to the problem of skeletal overgrowth the type of effort that has long been directed toward the correction of conditions which produce arrested growth is a fresh and valuable point of view emphasized by Chandler.1 A discussion of congenital enlargement of the extremities may profitably include both conditions involving the soft parts alone and conditions in which the entire extremity is affected. Three chief types of overgrowth can thus be discussed: congenital lymphedema (lymphangiectasis), congenital hemangiectatic hypertrophy of the extremities and Recklinghausen's neurofibromatosis. CONGENITAL LYMPHEDEMA (LYMPHANGIECTASIS) This condition is characterized by the presence at birth of nonpitting edema restricted to
American journal of diseases of children – American Medical Association
Published: Feb 1, 1939
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