1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080904020
This article is only available in the PDF format. Download the PDF to view the article, as well as its associated figures and tables.
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080785001pmid: 4580444
This article is only available in the PDF format. Download the PDF to view the article, as well as its associated figures and tables. Abstract AMERICAN otolaryngology owes a large debt to Dean M. Lierle who has just attained Emeritus status as the Executive Secretary-Treasurer of the American Board of Otolaryngology. He has occupied that office for 29 years and can be singled out as the one individual who has contributed more than anyone else in his generation to the advancement of graduate training in the specialty of otolaryngology. Dr. Lierle's professional life in otolaryngology began with his six years of residency training under Dr. Lee Wallace, Dean at Iowa, and was followed by a year of further training in Europe. This background and the numerous activities that Dr. Lierle developed in his professional life have enabled him to make his contributions. He became Head of the Department of Otolaryngology and Maxillofacial Surgery at Iowa, where he had trained, in 1929. In that year, the Board of Plastic Surgery was incorporated as a subsidiary of
Bridger, G. Patrick;Nassar, Victor H.
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080787002pmid: 4666420
Abstract Submucosal cancer extension through mucous glands occurs in the larynx. In a study of 45 cases, many lesions were too advanced to accurately state what had been the mode of spread. The majority of lesser lesions had clearly invaded according to the pattern predicted by the glandular theory. This theory offers an explanation for many clinical observations hitherto not completely understood—for example, the time honored dictum that supraglottic tumors tend not to invade the glottis but to penetrate the preepiglottic space. Anterior commissure tumors are difficult to treat because of their dissemination throughout the anterior subglottic mucous glands. Many new predictions are made; for example, on the behavior of ventricular and saccular tumors and on the distinction between cancers arising on the superior surface of the vocal cord and those showing mainly subglottic extension. References 1. McGavran MH, Bauer WC, Ogura JH: The incidence of cervical lymph node metastases from epidermoid carcinoma of the larynx . Cancer 14:55-66, 1961.Crossref 2. Work WP, Boyle WF: Cancer of the larynx . Laryngoscope 71:830-846, 1961.Crossref 3. Robbins R: Indications for radiation therapy in laryngeal cancer . Amer J Roentgen 83:21-24, 1960. 4. Baclesse F: Roentgenotherapy of carcinoma of the larynx . Clin Radiol 3:3-12, 1951. 5. Bocca E: II. Cancro del vestibolo laringeo . Arch Ital Otolaryng , (suppl 14) , p 65, 1953. 6. Pressman J, Simon MB, Monell C: Anatomical studies related to the dissemination of Cancer of the larynx . Trans Amer Acad Ophthal Otolaryng 64:628-638, 1960. 7. Clerf LH: Pre-epiglottic space, its relation to carcinoma of the epiglottis . Arch Otolaryng 40:177-179, 1944.Crossref 8. Ogura JH: Surgical pathology of cancer of the larynx . Laryngoscope 65:867-926, 1955.Crossref 9. Bocca E, Oreste P, Oreste M: Supraglottic surgery of the larynx . Ann Otol 77:1005-1026, 1968. 10. Kleinsasser O: Microlaryngoscopy and Endolaryngeal Microsurgery . Philadelphia, WB Saunders Co, 1968. 11. Broyles EN: The anterior commissure tendon . Ann Otol 52:342-345, 1943. 12. Tucker GF Jr, Smith R Jr: A histological demonstration of the development of laryngeal connective tissue compartments . Trans Amer Acad Ophthal Otolaryng 66:308-318, 1962. 13. Tucker GE Jr: Some clinical inferences from the study of serial sections . Laryngoscope 73:728-748, 1963.Crossref 14. Kirchner JA: One hundred laryngeal cancers studied by serial section . Ann Otol 78:689-709, 1969. 15. Bridger GP, Nassar VH: Carcinoma in situ, involving the laryngeal mucous glands. Arch Otolaryng, to be published. 16. Nassar VH, Bridger GP: Topography of the laryngeal mucous glands. Arch Otolaryng, to be published.
Anthony, Walter P.;Harrison, Clell W.
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080796003pmid: 4666421
Abstract The effects of both size and location of tympanic membrane perforations on hearing loss were studied. Using only those cases in which the air-bone gap was closed by myringoplasty, the variable of ossicular chain defects was controlled. Definite trends appeared in averages of the groups which tend to confirm two generalizations: (1) Effectiveness of the tympanic membrane decreases with loss of total area and (2) loss of membrane contact with the manubrium of the malleus affects its abiliy to move. A useful formula for predicting hearing loss on the basis of size and location of perforation is impossible because of wide variations of individual cases within any particular group. References 1. Cooper A: Observations of the effects which take place from the destruction of the membrana tympani of the ear (letter to Everard Home) , in Philosophical Transactions . London, W Bulmer & Co, 1800, pp 151-158. 2. Toynbee J: The Diseases of the Ear: Their Nature, Diagnosis, and Treatment . Philadelphia, Blanchard & Lea, 1860. 3. Treitel: Recent theories on sound conduction . Arch Otol 32:385-402, 1903. 4. Bingham WVD: The role of the tympanic mechanism in auditory . Psychol Rev 14:229-243, 1907.Crossref 5. Crowe SJ, Hughson W: Eine neue Methode zur Untersuchung der Psysiologic und Pathologic des Ohres . Z Hals Nas Ohrenheilk 30:65-76, 1931. 6. Crowe SJ, Hughson W: Experimental investigation of the physiology of the ear, using the method of Wever and Bray . Trans Amer Otol Cos 22:125-136, 1932. 7. Juers AL: Observations on bone conduction in fenestration cases . Ann Otol 57:28-41, 1948. 8. Minton JP: The dynamical function of the tympanic membrane and its associated ossicles . Proc Nat Acad Sci 11:439-445, 1925.Crossref 9. von Bekesy G: Uber die mechanisch-akustischen Vorgange beim Horen . Acta Otolaryng 27:281-296, 1939.Crossref 10. Pohlman AG: The reactions in the ear to sound . Ann Otol 50:363-378, 1941. 11. Lorente de No R, Harris AS: Experimental studies in hearing: II. The hearing loss after extirpation of the tympanic membrane . Laryngoscope 43:324-326, 1933. 12. Wever EG, Lawrence M, Smith KR: The middle ear in sound conduction . Arch Otolaryng 48:19-35, 1948.Crossref 13. Wever EG, Lawrence M: The transmission properties of the middle ear . Ann Otol 59:5-18, 1950. 14. Bordley JE, Hardy M: Effect of lesions of the tympanic membrane on the hearing acuity . Arch Otolaryng 26:649-657, 1937.Crossref 15. Payne MC Jr, Githler FJ: Effects of perforations of the tympanic membrane cochlear potentials . Arch Otolaryng 54:666-674, 1951.Crossref
Tos, Mirko;Bak-Pedersen, Kristian
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080801004pmid: 4666422
Abstract In a patient with mild chronic secretory otitis media, 488 abnormal mucous glands were found in the mucous membrane of the osseous eustachian tube and middle ear. Of these glands, 188 were active, 102 were transitional, and 198 were degenerated. The glands are described and their secretory capacity calculated. On the basis of these studies a new pathogenesis of chronic secretory otitis is presented and discussed. Owing to the influence of etiological factors, transformation of the epithelium with gland formation occurs. When the glands have completed their development they start forming mucus, which slowly accumulates in the middle ear. When the majority of the glands have started degenerating, the mucus production decreases and the disease improves spontaneously, provided that adhesive otitis has not arisen. The therapeutic program consists of (1) prevention of gland formation (2) intubation and (3) long-term follow-up of the patients. References 1. Politzer A: Lehrbuch der Ohrenheilkunde . Stuttgart, West Germany, Ferdinand Enke, 1878, p 298. 2. Bak-Pedersen K, Tos M: The mucous glands in chronic secretory otitis media . Acta Otolaryng , 72:14-27, 1971.Crossref 3. Friedmann I: The pathology of secretory otitis media . Proc Roy Soc Med 56:695-699, 1963. 4. Bendek GA: Histopathology of transudatory-secretory otitis media . Arch Otolaryng 78:33-38, 1963.Crossref 5. Sade J: Pathology and pathogenesis of serous otitis media . Arch Otolaryng 84:297-305, 1966.Crossref 6. Paparella MM, Lim DJ: Pathogenesis and pathology of the "idiopathic" blue ear drum . Arch Otolaryng 85:249-258, 1967.Crossref 7. Tos M: Mucous glands of the trachea in children. Quantitative studies . Anat Anz 126:146-160, 1970. 8. Tos M: Mucous glands of the trachea in man: Quantitative studies . Anat Anz 128:136-149, 1971. 9. Tos M: Distribution of mucous glands in the foetal eustachian-tube . Arch Klin Exp Ohr Nas Kehlkopfheilk 197:295-306, 1971.Crossref 10. Buch NH, Jorgensen MB: Eustachian tube and middle ear . Arch Otolaryng 79:472-480, 1964.Crossref 11. Polvogt L, Babb C: Histologie studies of the eustachian tube of individuals with good hearing . Laryngoscope 7:671-675, 1940. 12. Sade J, Weinberg J: Mucus production in the chronically infected middle ear . Ann Otol 78:148-155, 1969. 13. Friedmann I: The comparative pathology of otitis media—experimental and human . J Laryng 69:27-50, 1955.Crossref 14. Senturia BH, Carr CD, Ahlvin RC: Middle ear effusions: Pathologic changes of the mucoperiosteum in the experimental animal . Ann Otol 71:632-647, 1962. 15. Everberg G, Pulsen J, Rasmussen F: Den seorse otitis' klinik og behandling . Uges Laeg 130:1968-1973, 1968. 16. Tos M: Development of the tracheal glands in man . Acta Path Microbiol Scand , (suppl 185) , pp 1-130, 1966. 17. Patzelt V: Über die menschliche Epiglottis und die Entwicklung des Epithels in den Nachbargebieten . Z Anat Entwicklungsgesch 70:1-178, 1923.Crossref 18. Tos M: Development of the mucous glands in the human main bronchus . Anat Anz 123:376-389, 1968. 19. Tos M: Development of mucous glands in the human eustachian tube . Acta Otolaryng 70:340-350, 1970.Crossref 20. Sade J: Middle ear mucosa . Arch Otolaryng 84:137-143, 1966.Crossref 21. Ellefsen P, Tos M: Goblet cells in human trachea. Quantitative studies of a normal material. Anat Anz, to be published.
Whicker, James H.;Devine, Kenneth D.
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080812005pmid: 4592570
Abstract Based on the originality and accuracy of their scientific accomplishments, six anatomists emerge preeminent among the early contributors to the knowledge of laryngology. As long as 2,400 years ago, Hippocrates theorized about laryngeal function. In the succeeding centuries, Galen, da Vinci, Vesalius, Eustachius, and Magendie described laryngeal structures and innervation, established through anatomical dissection and experiment. Modern methods of evaluating glottic function often rely on information revealed centuries ago. References 1. Leonardo da Vinci . New York, Reynal & Co, 1956, p 399. 2. Duckworth WLH: Galen on Anatomical Procedures . Cambridge, England, Cambridge University Press, 1962. 3. O'Malley CD, Saunders JB de CM: Leonardo da Vinci on the Human Body . New York, Henry Schuman Inc, 1952, p 402. 4. Vesalius A: Icones Anatomicae . Germany, Universitatis Monacensis, 1934, p 28; 63. 5. Huard P: Léonardo de Vinci: Dessins anatomiques (anatomic artistique, descriptive et fonctionnelle) . Paris, Roger Dacosta, 1961, p 124. 6. Eustachius B: Explicatio Tabularum Anatomicarum . Rome, F Gonzagae, 1714, p 184. 7. Magendie F: A Summary of Philosophy . Baltimore, Edward J Coale & Co, 1822, p 120.
Weissman, Bruce W.;Panettiere, Frank
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080816006pmid: 4666423
Abstract Classical hemophilia results from a total or partial deficiency of AHG or antihemophiliac globulin (factor VIII). By the addition of this factor to the blood, the clotting mechanism will become normal. Modern management requires the use of concentrates of AHG to prevent circulatory overload previously resulting from massive transfusions. Close cooperation with the hematologist is required if elective surgery is to be safely performed. References 1. Strauss HS: Acquired circulating anticoagulants in hemophilia A . New Eng J Med 281: 866-873, 1969.Crossref 2. Dallman PR, Pool JG: Treatment of hemophilia with factor VIII concentrates . New Eng J Med 278:199-202, 1968.Crossref 3. Cooke JV, Holland PV, Shulman NR: Cryoprecipitate concentrates of factor VIII for surgery in hemophiliacs . Ann Intern Med 68:39-47, 1968.Crossref 4. Simson LR, Oberman HA, Penner JA: Clinical evaluation of cryoprecipitated factor VIII . JAMA 199:554-558, 1967.Crossref 5. Brinkhous KM, Shanbrom E, Roberts HR, et al: A new high-potency glycineprecipitated antihemophilic factor (AHG) concentrate . JAMA 205:613-617, 1968.Crossref 6. Soloway HB, Boreznak CE: Plasma fibrinogen levels following cryoprecipitate infusion . Transfusion 10:326-328, 1970.Crossref
Lundquist, Per-G.;Igarashi, Makoto;Wersäll, Jan;Alford, Bobby R.;Wright, William K.
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080820007pmid: 4125537
Abstract Experimental destruction of vestibular labyrinthine epithelia of the guinea pig with cryosurgery indicates that, with three minutes of treatment with −180 C (−292 F) and the cryoprobe applied at a precooled state, there is virtually no damage visible with light microscopy. However, with electron microscopy, regular signs of impaired metabolism are observed a few hours after treatment due to destroyed intramitochondrial membranes and formation of vacuoles in the cytoplasm. With the cryoprobe applied at room temperature and slowly cooled down to −180 C (−292 F), there is after 2½ hours a marked disintegration of the epithelial lining, presumably due to complete blocking of the cellular metabolism and cracking of the cytoplasm into fragments by ice crystal formation. It was possible with cryosurgery to experimentally produce a selective destruction of semicircular canal cristae with utricle, saccule, and cochlea remaining intact. References 1. Angell JJ: New developments in the ultrasonic therapy of Meniere's disease . Ann Roy Coll Surg 38:226-244, 1963. 2. Angell JJ: Meniere's disease: treatment with ultrasound . J Laryng 83:771-785, 1969.Crossref 3. Angell JJ: Ultrasound on the labyrinth , in Robbeh WF (ed): Proceedings of the Centennial Symposium, Manhattan Eye, Ear and Throat Hospital, Otolaryngology . St. Louis, CV Mosby Co Medical Publishers, 1969, vol 2, pp 136-139. 4. Arslan M: Ultrasonic destruction of the vestibular receptors , in Wolfson RJ: The Vestibular System and Its Diseases . Philadelphia, University of Pennsylvania Press, 1966, pp 515-526. 5. Sjöberg A, Stahle J, Johnsson S, et al: Treatment of Meniere's disease by ultrasonic irradiation . Acta Otolaryng , (suppl 178) , pp 1-86, 1963. 6. Cutt RA, Wolfson RJ, Ishiyama E, et al: Preliminary results with experimental cryosurgery of the labyrinth . Arch Otolaryng 82:147-158, 1965.Crossref 7. Cutt RA, Ishiyama E, Myers EN, et al: Histology of the monkey labyrinth following experimental cryosurgery . Ann Otol 77:275-285, 1968. 8. Cutt RA, Ishiyama E, Wolfson RJ: Experimental Cryosurgery on the labyrinth , in Proceedings of the Centennial Symposium, Manhattan Eye, Ear and Throat Hospital, Otolaryngology . vol 2, pp 120-129. 9. Wolfson RJ, Cutt RA, Ishiyama E, et al: Cryosurgery of the labyrinth: Preliminary report of a new surgical procedure . Laryngoscope 76:733-756, 1966.Crossref 10. Wolfson RJ, Cutt RA, Ishiyama E, et al: Cryosurgery for Meniere's disease . Laryngoscope 78:632-642, 1968.Crossref 11. Wolfson RJ, Cutt RA: Cryosurgery for Meniere's Disease , in Proceedings of the Centennial Symposium, Manhattan Eye, Ear and Throat Hospital, Otolaryngology . 1969, vol, 2, pp 130-135. 12. Wersäll J, Björkroth B, Flock A, et al: Sensory hair fusion in vestibular sensory cells after Gentamycin exposure: A transmission and scanning electron microscope study . Arch Klin Exp Ohr Nas Kehlkheilk 250:1-14, 1971.Crossref
Spooner, Thomas R.;Goode, Richard L.
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080833008pmid: 4666424
Abstract The phenomenon of hyperthermia sometimes causes new neurological findings in patients with multiple sclerosis. A series of 12 patients with multiple sclerosis revealed changes in the electronystagmograms when tested under hyperthermic conditions as compared to ten controls. References 1. Simons DJ: A note on the effect of heat and cold upon certain symptoms of multiple sclerosis . Bull Neurol Inst 6:385, 1937. 2. Collins RT: Transitory neurologic changes during hyperthermia . Bull Neurology Institute 7:291-296, 1938. 3. Nelson DA, Jeffreys WH, McDowell F: Effects of induced hyperthermia on some neurological diseases . Arch Neurol Psychiat 79:31-39, 1958.Crossref 4. Davis FA: The hot bath test in the diagnosis of multiple sclerosis . Mount Sinai Hosp J 33:280-282, 1966. 5. Dayal V, Tarantino L: Neuro-otologic studies in multiple sclerosis . Laryngoscope 76:1798-1809, 1966.Crossref
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080837009pmid: 4125538
Abstract In 50 patients 103 biopsies were taken during bronchoscopy from various sites of the trachea where the goblet cell density was determined quantitatively under various disease conditions. Twenty counts were done on each biopsy specimen on fields measuring 0.01768 sq mm for a total of 2,060 counts. The mean goblet cell density in normal tracheae was found to be 122 cells, in acute tracheobronchitis 141 cells, in mild chronic tracheobronchitis 145 cells, and in typical chronic tracheobronchitis 180 cells per field. There was a slight increase in goblet cell density with increasing consumption of cigarettes and a definite increase in chronic exposure to dust. In acute pulmonary diseases, such as pneumonia, bronchopneumonia, and pleuropneumonia, and in cancer of the lung, there was no increase in the goblet-cell density. References 1. Tos M: Development of the tracheal glands in man . Acta Path Microbiol Scand 68( (suppl 185) ):1-130, 1966. 2. Tos M: Distribution and situation of the mucous glands in the main bronchus of human foetus . Anat Anz 123:481-495, 1968. 3. Tos M: Topography of tracheal mucous glands in children . J Otolaryng 83:1073-1087, 1969. 4. Tos M: Anatomy of the tracheal mucous glands in man . Arch Otolaryng 92:132-137, 1970.Crossref 5. Reid L: Measurement of the bronchial mucus gland layer: A diagnostic yardstick in chronic bronchitis . Thorax 15:132-141, 1960.Crossref 6. Thurlbeck G, Angus GE, Paré JAP: Mucous gland hypertrophy in chronic bronchitis and its occurrence in smokers . Brit J Dis Chest 57:73-78, 1963.Crossref 7. Ellefsen P, Tos M: Goblet cells in human trachea: Quantitative studies of normal tracheae. Anat Anz, to be published. 8. Florey H, Carleton HM, Wells AO: Mucus secretion in the trachea . Brit J Exp Path 13:269-284, 1932. 9. Chang SM: Microscopic properties of whole mounts and secretions of human bronchial epithelium of smokers and nonsmokers . Cancer 10:1246-1262, 1957.Crossref 10. Reid L: Bronchial mucus production in health and disease , in Liebow AA, Smith DE (eds): The Lung . Baltimore, Williams & Wilkins Co, 1968, pp 87-108. 11. Lamb D, Reid L: Mitotic rates, goblet cell increase and histochemical changes in mucus in rat bronchial epithelium during exposure to sulphur dioxide . J Path Bact 96:97-111, 1968.Crossref 12. Tos M: Mucous glands of the trachea in children: Quantitative studies . Anat Anz 126:146-160, 1970. 13. Aurell G: Kolophonium-Chininhydrochloridgemische als Einschlussmittel fur sehr dicke Schnitte zu mikroskopischen Zwechen . Z Wiss Mikr 55:256-273, 1938. 14. Kessing SV: Mucous gland system of the conjunctiva . Acta Ophthalmol Scand , (suppl 95) , pp 1-133, 1968. 15. Bak-Pedersen K, Tos M: The mucous glands in chronic secretory otitis media. Acta Otolaryng Scand, to be published.
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080846010pmid: 4666425
Abstract Five patients demonstrated a sensorineural hearing defect following aural barotrauma. Preincident and postincident audiograms show the extent of impairment to vary from a high frequency loss to a total sensorineural deafness. The men involved were all Navy divers and they all experienced difficulty in clearing their ears on descent. The terminology recommended for this condition is "inner ear barotrauma," to differentiate it from other forms of barotrauma and from the involvement of the eighth nerve in decompression sickness. References 1. Almour: Industrial otology in caisson workers . N Y State J Med 42:779-785, 1942. 2. Boot GW: Caisson workers' deafness . Ann Otol 22:1121-1132, 1913. 3. Shilling CW, Everly IA: Auditory acuity in submarine personnel . US Naval Med Bull 40:664-686, 1942. 4. Haines HL, Harris JD: Aerotitis media in submariners . Ann Otol 55:347-371, 1946. 5. Taylor GD: The otolaryngologic aspects of skin and scuba diving . Laryngoscope 69:809-858, 1959.Crossref 6. Rawlins JSP, quoted by Coles RAA, Knight JS: Aural and audiometry survey of qualified divers and submarine escape training tank instructors . MCR Report RNP 61:1011, 1961. 7. Coles RAA, Knight JJ: Aural and audiometry survey of qualified divers and submarine escape training tank instructors . MRC Report RNP 61:1011, 1961. 8. MacFie D: ENT problems of diving . Med Serv J Canad 20:845-851, 1964. 9. Demand F, Fredenwucci P, Appoix A: Hyperbaric treatment of sudden deafness and inner ear deafness resulting from barotrauma . Bull MEDSUBHYP Soc Franc Med 1:1969. 10. Eichel BS, Landes BS: Sensorineural hearing loss caused by skin diving . Arch Otolaryng 92:128-131, 1970.Crossref
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080854011pmid: 4666426
Abstract Pure cochlear otosclerosis is slowly becoming recognized as a serious cause of sensorineural hearing loss comparatively early in adult life. The importance of correct identification is stressed by the possibility that fluoride treatment may effectively halt its advance. References 1. Siebenmann F: Multiple Spongiosierung der Labyrinthkapsel als Sectionsbefund bei einem Fall von progressiver Schwerhorigkeit . Z Ohrenheilk 34: 356-374, 1899. 2. Krepuska G, Krepuska I: Otology: Magyar Orvosi Koenyvkiado . Budapest, Tarsulat, 1936. 3. Guild SR: Does otosclerosis cause cochlear nerve degeneration . Trans Amer Acad Ophthal Otolaryng 57:356-365, 1953. 4. Glorig A, Gallo R: Comments on sensori-neural hearing loss in otosclerosis in Schuknecht HF (ed): Otosclerosis . Boston, Little Brown & Co, 1962, pp 63-78. 5. Kelemen G, Linthicum FH Jr: Labyrinthine otosclerosis . Acta Otolaryng , (suppl 253) , 1969. 6. Linthicum FH Jr: Correlation of sensorineural hearing impairment and otosclerosis . Ann Otol 75: 512, 1966. 7. Nager GT: Histopathology of otosclerosis . Arch Otolaryng 89:341-363, 1969.Crossref 8. Ruedi L: Histopathology of Sensorineural Degeneration and Other Inner Ear Changes in Otosclerosis , in Henry Ford Hospital International Symposium: Otosclerosis . Detroit, Little Brown & Co, 1962. 9. Brunner S, Rovsing H, Jensen J: Tomographic changes in otosclerosis . Acta Radiol 4:632-638, 1966.Crossref 10. Derlacki EL, Valvassori G: Clinical and radiological diagnosis of labyrinthine otosclerosis . Trans Amer Laryngol Rhinol Otol Soc 381-395, 1965. 11. Hoople GD, Basch RI: Radiographic findings in cochlear otosclerosis . Ann Otol 75:688-697, 1966. 12. Jensen J, Rovsing H, Brunner S: Tomography of the inner ear in otosclerosis . Brit J Radiol 39: 669-672, 1966.Crossref 13. Portmann M, Guillen G: Radiodiagnostic en Otologie . Paris, Masson et Cie, 1959. 14. Valvassori GE: Otosclerosis: A new challenge to roentgenology . Amer J Roentgen 94:566-575, 1965. 15. Shambaugh GE Jr: Sodium fluoride for inactivation of the otosclerotic lesion . Arch Otolaryng 89:381-382, 1969.Crossref 16. Shambaugh GE Jr: Otosclerosis in otolaryngology , in Otosclerosis . New York, Harper & Row Publishers Inc, 1959, p 8.
Goodwin, Maurice R.;Wolfe, Asher
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080860012pmid: 4666427
Abstract A patient who had received intense electrical shock while on the job sustained an injury to his hearing mechanism that would appear to include the neural pathways. Indeed, the slight loss for pure-tone threshold on initial examination would tend to indicate only minimal cochlear pathology. References 1. Gabrielli L: Unusual clinical picture of intermittent deafness in a subject struck by lightning . Otorinolaring Ital 31:79-90, 1962. 2. Jemmi A: Cochleovestibular lesions caused by electrical energy . Otorinolaring Ital 34:232-265, 1965. 3. Miszke A: Lightning injuries in laryngology . Otolaryng Pol 16:527-534, 1962. 4. Mounier-Kuhn P: Clinical study of lesions of the auditory apparatus caused by electricity . Rev Otoneuroopthal 35:165-176, 1963. 5. Piatti A: Bilateral word deafness and vestibular damage caused by electrocution . Arch Ital Otol 74: 475-479, 1963. 6. Tolnay S: Lightning injury of the acoustic organ . Orv Hetil 108:696-698, 1967. 7. Fowler EP: Marked deafness areas in normal ears . Arch Otolaryng 8:151-155, 1928.Crossref 8. Jerger J: Bekesy audiometry in analysis of auditory disorders . J Speech Hearing Res 3:275-287, 1962.
Kamerer, Donald B.;Dickinson, John T.;Cipcic, Joseph A.
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080864013pmid: 4666428
Abstract Epidural hemorrhage is a rare complication of otitis media. There are probably no satisfactory criteria to enable one to diagnose epidural hemorrhage preoperatively. The suspicion of intracranial complications resulting from middle ear and mastoid disease justifies surgical intervention. References 1. Schneider R, Hegarty W: Extradural hemorrhage as a complication of otological and rhinological infections . Ann Otol 60:197, 1951. 2. Clein LF: Extradural hematoma associated with middle ear infection . Canad Med Assoc J 102:1183-1184, 1970. 3. Novaes V, Gorbitz C: Extradural hematoma complicating middle ear infection . J Neurosurg 23: 352-353, 1965.Crossref
Hagadorn, Bruce;Smith, Howard W.;Rosnagle, Robert S.
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080868014pmid: 4666429
Abstract Foreign bodies of the hypopharynx may result in serious complications several weeks after their removal. Persistent symptoms should alert the physician to continuing infection. Osteomyelitis of the cervical spine has been seen as a complication of a foreign body in the hypopharynx which had penetrated the posterior pharyngeal wall. References 1. Makins GH, Abbott FL: On acute primary osteomyelitis of the vertebrae . Ann Surg 23:510-539, 1896.Crossref 2. Wilensky AO: Osteomyelitis of the vertebrae . Ann Surg 89:561-570, 731-747, 1929.Crossref 3. Henson SW Jr, Coventry MB: Osteomyelitis of the vertebrae as the result of infection of the urinary tract . Surg Gynec Obstet 102:207-214, 1956. 4. Hutton PW: Acute osteomyelitis of the cervical spine with epidural abscess . Brit Med J 1:153-154, 1956.Crossref 5. Ablin G, Erickson TC: Osteomyelitis of the cervical vertebrae secondary to urinary tract infection . J Neurosurg 15:455-459, 1958.Crossref 6. Durity F, Thompson GB: Localized cervical extradural abscess . J Neurosurg 28:387-390, 1968.Crossref 7. Jackson FE, et al: Quadriplegia caused by involvement of cervical spine with Coccidioides immitis . J Neurosurg 21:512-515, 1964.Crossref 8. Stone DB, Bonfiglio M: Pyogenic vertebral osteomyelitis . Arch Intern Med 112:491-500, 1963.Crossref 9. Finch PG: Staphylococcal osteomyelitis of spine in a baby aged 3 weeks . Lancet 2:134-135, 1947.Crossref 10. Wiley AM, Trueta J: The vascular anatomy of the spine and its relationship to pyogenic vertebral osteomyelitis . J Bone Joint Surg 41:796-809, 1959.
Sholehvar, Javad;Hunsicker, Robert C.;Stool, Sylvan E.
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080871015pmid: 4666430
Abstract The occasional postadenotonsillectomy bleeder may require ligation of the external carotid artery. This case report demonstrates the need for arresting flow through the branches of the carotid in order to assure hemostasis. Carotid arteriography is suggested as a valuable tool in the management of selected cases of tonsillar hemorrhage. References 1. Proctor DF: Tonsils and Adenoids in Children . Springfield, Ill, Charles C Thomas Publisher, 1966. 2. Breson K, Diepeveen J: Dissection tonsillectomy . J Laryngol 83:601-608, 1969.Crossref 3. Talbot H: Adenotonsillectomy: Technique and postoperative care . Laryngoscope 75:1877-1891, 1965.Crossref 4. Vital Statistics of the United States. Department of Health, Education, and Welfare, vol 2. Mortality, part A, 1965, through 1967. 5. Grant JCB: Fig 513.2 , in An Atlas of Anatomy . Baltimore, Williams & Wilkins Co, 1956. 6. Pratt LW, Root JA: Catastrophic post-tonsillectomy secondary hemorrhage . J Maine Med Assoc 51:7-12, 1960. 7. Hollinshead WH: Fig 285 , in Anatomy for Surgeons . New York, Paul B Hoeber Inc, Medical Bk Dept of Harper & Row Publishers Inc, 1958, vol 1, p 476. 8. Dorrance GM: Ligation of the great vessels of the neck . Ann Surg 99:721-742, 1934.Crossref 9. Lang EK, Hahn C, Luvos TJ: Arteriographic demonstration of internal-external carotid anastamoses and their correlation to RISA circulation studies . Radiology 83:632-639, 1964.Crossref 10. Tindall GT, Odom GL, Dillon ML, et al: Direction of blood flow in the internal and external carotid arteries following occlusion of the ipsilateral common carotid artery . J Neurosurg 20:985-994, 1963.Crossref 11. Gardner JF: Sutures and disasters in tonsillectomy . Arch Otolaryng 88:551-557, 1968.Crossref
SATALOFF, JOSEPH;FECHNER, ROBERT E.
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080874016
Abstract PATHOLOGIC QUIZ CASE 1 Philip L. Martin, MD, Anchorage, AlaskaA 34-year-old Eskimo woman had had a slowly enlarging parotid mass on the right side for one year. She denied experiencing facial pain, weakness, or numbness during this length of time. She also denied experiencing ocular or oval dryness. She did relate a history of pulmonary tuberculosis 13 years previously. Physical examination demonstrated a firm, movable, nontender, 2×3 cm mass in the superficial portion of the right parotid. Shotty cervical adenopathy was present bilaterally. There was no evidence of xerophthalmia or xerostomia. Results of a nasopharyngeal examination were normal. A sialogram depicted the parotid mass. Ductal architecture was medially and inferiorly displaced. At surgery, an easily dissectable mass was excised from the superficial portion of the parotid (Fig 1 to 3). Peripheral branches of the facial nerve were not involved in this lesion. A contiguous lymph node was taken with References 1. Gravanis MB, et al.: Malignant histopathologic counterpart of the benign lymphoepithelial lesion . Cancer 26: 1332-1342, 1970.Crossref 2. Rosalki SB, McGee LE: Meningioma presenting as nasopharyngeal tumor: Report of two cases . J Laryng 76:133-139, 1962.Crossref 3. McGavran MH, Biller HF, Ogura JH: Primary intranasal meningioma . Arch Otolaryng 93:95-97, 1971.Crossref
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080878017pmid: 4666431
Abstract To the Editor.—Freeman and Edmonds reported five cases of "Inner Ear Barotrauma." In each case persistent sensorineural hearing loss followed diving episodes. All five patients were trained Navy divers with preincident and postincident audiograms. The authors point out clearly that in none of the cases could the inner ear lesion be attributed to decompression sickness or to pulmonary barotrauma. Thus, nitrogen embolization has been ruled out by the authors as a possible etiologic factor. All five divers had difficulty in performing Valsalva maneuvers on the affected side, but in only one case was there otoscopic evidence of middle ear involvement. In several cases vestibular symptoms were also present. The hearing losses were either immediate or delayed by a number of hours. Barotrauma seems a likely etiologic factor and "inner ear barotrauma" is a reasonable descriptive term. In the discussion of mechanisms, the authors discuss the possible effect of pressure References 1. Goodhill V: Sudden deafness and round window rupture . Laryngoscope 81:1462-1474, 1971.Crossref
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080879018
This article is only available in the PDF format. Download the PDF to view the article, as well as its associated figures and tables. Abstract Announcement.—The dates for the 1972 examination of the American Board of Otolaryngology are Oct 21-26, 1972. They will be held at the Palmer House in Chicago. Congress.—The dates of the 1972 Congress of the International Society of Audiology have been changed from Sept 26-30, 1972, to Oct 3-7, 1972, because of a conflict with the meeting of the American Academy of Ophthalmology and Otolaryngology. Announcement.—The Deafness Research Foundation announces that applications for research grants for the calendar year 1973 must be submitted prior to Aug 15, 1972, for consideration by the Scientific Review Committee. For further information and application forms, write to The Deafness Research Foundation, 366 Madison Ave, New York 10017. Meeting.—The Annual Midsummer Meeting, United States Section, International College of Surgeons, will be held July 5 through 10, 1972, at the Sagamore Hotel, Bolton Landing, Lake George, NY. An outstanding full three-day program has
1972 Archives of Otolaryngology
doi: 10.1001/archotol.1972.00770080881019
This article is only available in the PDF format. Download the PDF to view the article, as well as its associated figures and tables.