Archives of Ophthalmology

Blodi, Frederick C.

1980 Archives of Ophthalmology

doi: 10.1001/archopht.1980.01020040411001

This article is only available in the PDF format. Download the PDF to view the article, as well as its associated figures and tables. Abstract San Francisco is the host city for the XXIV International Congress of Ophthalmology, which will convene Oct 31 through Nov 5, 1982. The history of these meetings goes back 125 years to the first congress held in Brussels. Their purpose was and continues to be to bring together ophthalmologists from around the world to exchange new ideas and information to advance the treatment of eye diseases. The board of directors of the American Academy of Ophthalmology, as part of their decision to organize and sponsor the congress, agreed to combine the annual meeting of the American Academy with the international congress. This effort will probably produce the largest gathering of ophthalmologists in history. Attendance is expected to reach 20,000 to 25,000. The Organizing Committee has requested abstracts of scientific papers, exhibits, or films be mailed so that they are received by Dec 15, 1981. Requests for abstract submission forms should

Heijl, Anders;Drance, Stephen M.;Douglas, Gordon R.

1980 Archives of Ophthalmology

doi: 10.1001/archopht.1980.01020040412002pmid: 7425915

Abstract • The ability of fully automatic computerized perimetry to detect early glaucomatous field damage was compared with that of careful static and kinetic manual perimetry in a clinical study on 104 patients, 51 of whom had early glaucomatous field defects, 20 of whom were glaucoma suspects with no field defects, and 33 of whom were normal. The automatic perimeter used was the COMPETER automatic perimeter employing actual threshold measurements. The interpretation of the automatic fields followed a set of predetermined criteria. Fifty-one eyes had defects in the manual charts, 48 (94%) of which were detected by automatic perimetry using the central test point pattern of the perimeter. Two (4%) fields thought to be normal after manual perimetry were correctly found to be abnormal by the automatic perimeter, which yielded four (8%) false-positives in the normal fields. By using a different set of criteria for the interpretation of the automatic fields, the sensitivity could be increased to 98% of these early defects, but at the cost of 22% false-positives. References 1. Heijl A: Automatic perimetry in glaucoma visual field screening: A clinical study . Albrecht Von Graefes Arch Klin Exp Ophthalmol 200:21-37, 1976.Crossref 2. Heijl A, Krakau CET: An automatic perimeter for glaucoma visual field screening and control: Construction and clinical cases . Albrecht Von Graefes Arch Klin Exp Ophthalmol 197:13-23, 1975.Crossref 3. Armaly MF: Selective perimetry for glaucomatous defects in ocular hypertension . Arch Ophthalmol 87:518-524, 1972.Crossref 4. Heijl A, Krakau CET: A note on fixation during perimetry . Acta Ophthalmol 55:854-861, 1977.Crossref 5. Heijl A: Computer test logics for automatic perimetry . Acta Ophthalmol 55:837-853, 1977.Crossref 6. Krakau CET: Aspects on the design of an automatic perimeter . Acta Ophthalmol 56:389-405, 1978.Crossref

Quigley, Harry A.;Miller, Neil R.;George, Terry

1980 Archives of Ophthalmology

doi: 10.1001/archopht.1980.01020040416003pmid: 7425916

Abstract • To evaluate the usefulness of the retinal nerve fiber layer (NFL) appearance in the estimation of glaucomatous optic nerve damage, we examined 335 eyes of normal, glaucoma suspect, or glaucomatous patients. In eyes with field loss, 84% had NFL atrophy. In normal eyes, NFL atrophy was suspected in 3%. In glaucoma suspects, 13% of eyes had NFL defects. Defects in glaucoma suspect eyes were more often localized, compared with the diffuse atrophy found in eyes with visual field loss. The NFL abnormalities were seen in areas of the retina corresponding to the location of visual field defects with a high accuracy. The NFL examination is as sensitive in detecting eyes with field loss as the optic disc configuration and may help to identify which eyes are suffering optic nerve damage prior to field loss. References 1. Armaly MF: Ocular pressure and visual fields: A ten-year follow-up study . Arch Ophthalmol 81:25-40, 1969.Crossref 2. Graham PA: The definition of pre-glaucoma, a prospective study . Trans Ophthalmol Soc UK 88:153-165, 1968. 3. Perkins ES: The Bedford glaucoma survey: I. Long-term follow-up of borderline cases . Br J Ophthalmol 57:179-185, 1973.Crossref 4. Wilensky JT, Podos SM: Prognostic parameters in primary open angle glaucoma , in Anderson DR, Drance SM, Galin MA, et al (eds): Symposium on Glaucoma . St Louis, CV Mosby Co, 1975, pp 7-30. 5. Hart WM Jr, Yablonski M, Kass MA, et al: Multivariate analysis of the risk of glaucomatous visual field loss . Arch Ophthalmol 97:1455-1458, 1979.Crossref 6. Hoyt WF: Ophthalmoscopy of the retinal nerve fiber layer in neuroophthalmologic diagnosis . Aust J Ophthalmol 4:14-34, 1976.Crossref 7. Hoyt WF, Frisen L, Newman NM: Fundoscopy of the nerve fiber layer defects in glaucoma . Invest Ophthalmol 12:814-829, 1973. 8. Sommer A, Miller NR, Pollack I, et al: The nerve fiber layer in the diagnosis of glaucoma . Arch Ophthalmol 95:2149-2156, 1977.Crossref 9. Sommer A, Pollack I, Maumenee AE: Optic disc parameters and onset of glaucomatous field loss: I. Methods and progressive changes in disc morphology . Arch Ophthalmol 97:1444-1448, 1979.Crossref 10. Sommer A, Pollack I, Maumenee AE: Optic disc parameters and onset of glaucomatous field loss: II. Static screening criteria . Arch Ophthalmol 97:1449-1454, 1979.Crossref 11. Rock WJ, Drance SM, Morgan RW: A modification of the Armaly visual field screening technique for glaucoma . Can J Ophthalmol 6:283-292, 1971. 12. Miller NR, George T: Monochromatic (redfree) photography and ophthalmoscopy of the peripapillary retinal nerve fiber layer . Invest Ophthalmol Vis Sci 17:1121-1124, 1978. 13. Schwartz JT: Methodologic differences and measurement of cup-disc ratio: An epidemiologic assessment . Arch Ophthalmol 94:1101-1105, 1976.Crossref 14. Kirsch RE, Anderson DR: Identification of the glaucomatous disc . Trans Am Acad Ophthalmol Otolaryngol 77:143-156, 1973. 15. Susanna R, Drance SM, Douglas GR: Disc hemorrhages in patients with elevated intraocular pressure: Occurrence with and without field changes . Arch Ophthalmol 97:284-285, 1979.Crossref 16. Hitchings RA, Spaeth GL: The optic disc in glaucoma: I. Classification . Br J Ophthalmol 60:778-785, 1976.Crossref 17. Hoskins HD, Gelber EC: Optic disk topography and visual field defects in patients with increased intraocular pressure . Am J Ophthalmol 80:284-290, 1975. 18. Drance SM: Correlation between optic disc changes and visual field defects in chronic openangle glaucoma . Trans Am Acad Ophthalmol Otolaryngol 81:224-226, 1976. 19. Harbin TS, Podos SM, Kolker AE, et al: Visual field progression in open-angle glaucoma patients presenting with monocular field loss . Trans Am Acad Ophthalmol Otolaryngol 81:253-257, 1976. 20. Quigley HA, Green WR: The histology of human glaucoma cupping and optic nerve damage: Clinicopathologic correlation in 21 eyes. Ophthalmology, to be published. 21. Arden GB, Jacobson JJ: A simple grating test for contrast sensitivity: Preliminary results indicate value in screening for glaucoma . Invest Ophthalmol Vis Sci 17:23-32, 1978. 22. Atkin A, Bodis-Wollner I, Wolkenstein M, et al: Abnormalities of central contrast sensitivity in glaucoma . Am J Ophthalmol 88:205-211, 1979.

Sinclair, Stephen H.;Pelham, Valerie;Giovanoni, Richard;Regan, Charles D. J.

1980 Archives of Ophthalmology

doi: 10.1001/archopht.1980.01020040424004pmid: 7425917

Abstract • A mydriatic used for outpatient indirect ophthalmoscopy should produce prompt, maximal, transient mydriasis after a single instillation. Cycloplegia or systemic side effects should be minimal. A solution of tropicamide with phenylephrine hydrochloride seems to achieve this most effectively. In a general retina clinic population, the percentage of pupils dilated, the degree of dilation, and the resistance of the dilation to intense illumination were used to evaluate various concentrations of the drugs. Eight-tenths percent tropicamide with 5% phenylephrine adequately dilated 98.8% of the eyes, among which 25% had dark irides and 9% were receiving miotic therapy. Reduction of the concentration of either component produced less adequate mydriasis; an increase of the tropicamide concentration resulted in a saturated solution with drug precipitate forming on storage and a less adequate dilation. With the optimal combination, 1 drop adequately dilates the general population; 2 drops appear no more effective than 1. References 1. Duke-Elder S: The Foundation of Ophthalmology: Hereditary and Pathology, Diagnosis and Therapeutics . St Louis, CV Mosby Co, 1964, vol 7, pp 543-586. 2. Barber RF, Smith WO: A comparative study of mydriatic and cycloplegic agents: In human subjects without eye disease . Am J Ophthalmol 44:617-622, 1957. 3. Gambill HD, Ogle KN, Kearns TP: Mydriatic effect of four drugs determined with pupillograph . Arch Ophthalmol 77:740-746, 1967.Crossref 4. Havener WH: Ocular Pharmacology , ed 3. St Louis, CV Mosby Co, 1974, pp 232-255. 5. Gettes BC, Belmont O: Tropicamide: Comparative cycloplegic effects . Arch Ophthalmol 66:336-340, 1961.Crossref 6. Milder B: Tropicamide as a cycloplegic agent . Arch Ophthalmol 66:70-72, 1961.Crossref 7. Solosko D, Smith RB: Hypertension following 10% phenylephrine ophthalmic . Anesthesiology 36:187-189, 1972.Crossref 8. Mays WL: Statistics for the Social Sciences . New York, Holt Rinehart & Winston Inc, 1973, pp 740-742. 9. Kim JM, Stevenson CE, Mathewson HS: Hypertensive reactions to phenylephrine eyedrops in patients with sympathetic denervation . Am J Ophthalmol 85:862-868, 1978. 10. McReynolds WU, Havener WH, Henderson JW: Hazards of the use of sympathomimetic drugs in ophthalmology . Arch Ophthalmol 56:176-179, 1956.Crossref 11. Fraunfelder FT: Interim report: National registry of possible drug-induced ocular side effects . Ophthalmology 86:126-130, 1979.Crossref

Morin, J. Donald;Bryars, John H.

1980 Archives of Ophthalmology

doi: 10.1001/archopht.1980.01020040427005pmid: 7425918

Abstract • Of 51 patients with congenital glaucoma, 13 had unilateral and 38 bilateral disease. Pressure was controlled at 8 to 24 mm Hg in 76 of the 89 affected eyes. Forty-one of these 76 eyes had visual acuity poorer than 6/15 (20/50); optic nerve damage as shown by visual field testing was the cause in 20, and medial opacities, including irregular corneal astigmatism, was the cause in 15. In only six eyes did the poor vision result from anisometropic amblyopia. References 1. Bryars JH: An Investigation of the Visually Handicapped Children in Northern Ireland, thesis, Belfast, 1977. 2. Richardson KT, Shaffer RN: Optic-nerve cupping in congenital glaucoma . Am J Ophthalmol 62:507-509, 1966. 3. Richardson KT Jr, Ferguson WJ Jr, Shaffer RN: Long-term functional results in infantile glaucoma . Trans Am Acad Ophthalmol Otolaryngol 71:833-836, 1967. 4. Adler FH, in discussion, Richardson KT Jr, Ferguson WJ Jr, Shaffer RN: Long-term functional results in infantile glaucoma . Trans Am Acad Ophthalmol Otolaryngol 71:836-837, 1967. 5. Scheie HG: The management of infantile glaucoma . Arch Ophthalmol 62:35-54, 1959.Crossref 6. Morin JD, Merin S, Sheppard RW: Primary congenital glaucoma: A survey . Can J Ophthalmol 9:17-28, 1974. 7. Maloley MA: The long-term results of occlusion therapy for amblyopia. Read before the annual meeting of the Canadian Ophthalmological Society (Orthoptics Section), Calgary, Alberta, June 5-7, 1977.

Mindel, Joel S.;Tavitian, Henry O.;Smith, Harry;Walker, Ethel C.

1980 Archives of Ophthalmology

doi: 10.1001/archopht.1980.01020040429006pmid: 6893544

Abstract • In age-, race-, and sex-matched groups, the relative abilities of medrysone, fluorometholone, and dexamethasone phosphate to elevate intraocular pressure were compared in a masked manner. Compliance was controlled by drops being given by nursing personnel. The 18 male hospitalized volunteers received commercial preparations of one of the corticosteroids to each eye four times a day for six weeks. The relative ability of medrysone, fluorometholone, and dexamethasone phosphate to raise IOP was 1, 4, and 7.6, respectively. References 1. Mindel JS, Goldberg J, Tavitian HO: Similarity of the intraocular pressure response to different corticosteroid esters when compliance is controlled . Ophthalmology 86:99-107, 1979.Crossref 2. Drance SM, Scott S: A comparison of action of dexamethasone and medrysone on human intraocular pressure . Can J Ophthalmol 3:159-161, 1968. 3. Kazantzis M: Etude comparative de l'action de la dexamethasone et de la medrysone sur la tension oculaire et la resistance a l'ecoulement . Ann Oculist 206:127-132, 1973. 4. Podos SM, Krupin T, Asseff C, et al: Topically administered corticosteroid preparations: Comparison of intraocular pressure effects . Arch Ophthalmol 86:251-254, 1971.Crossref 5. Kitazawa Y: Increased intraocular pressure induced by corticosteroids . Am J Ophthalmol 82:492-495, 1976. 6. Becker B: Intraocular pressure response to topical corticosteroids . Invest Ophthalmol 4:198-205, 1965.

Talusan, Eduardo D.;Schwartz, Bernard;Wilcox, Lloyd M.

1980 Archives of Ophthalmology

doi: 10.1001/archopht.1980.01020040431007pmid: 7425919

Abstract • A comparison of two or more disc fluorescein angiograms performed on separate occasions was done on 60 eyes of normal subjects and patients with ocular hypertension, primary open-angle glaucoma, and low-tension glaucoma. Clinically stable patients did not show any change in their disc angiographic filling patterns. Eyes that developed new visualfield defects with increased disc cupping and pallor correspondingly showed new absolute filling defects or areas of hypofluorescence. In those with established field defects, however, further changes in the visual field occurred without obvious changes in the disc fluorescein filling defect. Surgical lowering of intraocular pressure with or without decrease in disc cupping and pallor did not result in visible improvement of the disc angiographic pattern. Thus, the development of new visual-field defects is associated with changes of the circulation of the optic disc. References 1. Schwartz B: Primary open angle glaucoma , in Duane TD (ed): Clinical Ophthalmology . Hagerstown, Md, Harper & Row Publishers, 1976, pp 10-22. 2. Laatikainen L, Mantyla P: Effects of a fall in the intraocular pressure level on the peripappilary fluorescein angiogram in chronic open angle glaucoma . Acta Ophthalmol 52:625-633, 1974.Crossref 3. Spaeth GL: Fluorescein angiography: Its contribution towards understanding the mechanisms of visual loss in glaucoma . Trans Am Ophthalmol Soc 73:491-553, 1975. 4. Hayreh SS, Walker WM: Fluorescent fundus photography in glaucoma . Am J Ophthalmol 63:982-989, 1967. 5. Hitchings RA, Spaeth GL: Fluorescein angiography in glaucoma , in Cant CJ (ed): Vision and Circulation . London, Henry Kimpton Medical Publisher & Bookseller, 1976, pp 201-216. 6. Schwartz B, Rieser JC, Fishbein SL: Fluorescein angiographic defects of the optic disc in glaucoma . Arch Ophthalmol 95:1961-1974, 1977.Crossref 7. Fishbein SL, Schwartz B: Optic disc in glaucoma: Topography and extent of fluorescein filling defects . Arch Ophthalmol 95:1975-1978, 1977.Crossref 8. Tsukahara S, Nagatek S, Sugaya M, et al: Visual field defects, cup-disc ratio and fluorescein angiography in glaucomatous optic atrophy . Adv Ophthalmol 35:73-93, 1978. 9. Loebl M, Schwartz B: Fluorescein angiographic defects of the optic disc in ocular hypertension . Arch Ophthalmol 95:1980-1984, 1977.Crossref 10. Talusan ED, Schwartz B: Specificity of fluorescein angiographic defects of the optic disc in glaucoma . Arch Ophthalmol 95:2166-2175, 1977.Crossref 11. Tsukahara S: Hyperpermeable disc capillaries in glaucoma . Adv Ophthalmol 35:65-72, 1978. 12. Drance SM, Begg IS, Goldman H: Fluorescein angiography in the evaluation of focal circulatory ischemia of the optic nerve head in relation to the arcuate scotoma in glaucoma . Can J Ophthalmol 7:68-74, 1972. 13. Ben-Sira I, Loebl M, Schwartz B, et al: In vivo measurements of diffusion of fluorescein in the human optic disc nerve tissue , in Documenta Ophthalmologica Proceedings Series: International Symposium on Fluorescein Angiography . The Hague, W Junk Publishers, 1976, pp 311-314. 14. Spaeth GL: The Pathogenesis of Nerve Damage in Glaucoma: Contribution of Fluorescein Angiography . New York, Grune & Stratton Inc, 1977. 15. Hayreh SS: Color and fluorescence of the optic disc . Ophthalmologica 165:100-108, 1975.Crossref

Ziemianski, Matthew C.;Lee, King Y.;Sabates, Felix N.

1980 Archives of Ophthalmology

doi: 10.1001/archopht.1980.01020040440008pmid: 7191701

Abstract • A 15-year-old boy was seen initially with decreased vision in his left eye. Ophthalmoscopic examination showed numerous pigmented subretinal tracts with traction lines radiating to the fovea. Peripheral examination disclosed a maggot in the superior nasal quadrant of the vitreous. There was a hemorrhage emerging from the exit wound in the retina. Removal of the maggot was not indicated because there was no inflammation. References 1. Chandler AC: Diptera fly maggots and myiasis , in Introduction to Parasitology . New York, John Wiley & Sons Inc, 1956, pp 742-771. 2. Anderson WB: Ophthalmomyiasis interna: Case report and review of the literature . Trans Am Acad Ophthal Otolaryngol 39:218-231, 1934. 3. Gass JDM, Lewis RA: Subretinal tracks in ophthalmomyiasis . Arch Ophthalmol 94:1500-1505, 1976.Crossref 4. Adda V: Uber einen fall von Ophthalmomyiasis . Ophthalmologica 118:1002-1009, 1949.Crossref 5. Hervouet F, Ballard, Donnot, et al: Un cas de myase "hypoderma bovis" a localisation intra-camerienne . Bull Soc Ophtalmol Fr 2:129-133, 1960. 6. Godde-Jolly D, Brumpt ML, Stofft P: Un cas d'ophthalmomyase "hypoderma bovis" de la chambre anteriere . Bull Soc Ophtalmol Fr 2:48-56, 1966. 7. Borgstrom F: Experimental Cochliomyia americana infestations . Am J Trop Med 18:395-411, 1938. 8. Fitzgerald CR, Rubin ML: Intraocular parasite destroyed by photocoagulation . Arch Ophthalmol 91:162-164, 1974.Crossref

Adam, Giora;Schwartz, Bernard

1980 Archives of Ophthalmology

doi: 10.1001/archopht.1980.01020040442009pmid: 7425920

Abstract • One hundred seventy-one optic disc fluorescein angiograms of glaucomatous, ocular hypertensive, and normal patients were studied for the occurrence of areas of hypofluorescence or absolute filling defects in the rim, wall, and floor of the optic cup using disc stereo photographs compared with the fluorescein angiogram. The frequencies of involvement of the rim, wall, and floor of the cup by filling defects were determined. The wall of the cup has a greater percentage of filling defects in subjects with glaucoma than in ocular hypertensive or in normal subjects. The floor of the cup has more filling defects in normal subjects than in those with glaucoma. The involvement of the wall of the cup increases with the degree of visual-field loss in glaucoma. We suggest that, in glaucoma, the effect of ocular pressure is greater on the wall of the cup than on the floor of the cup. References 1. Hayreh SS, Walker WM: Fluorescent fundus photography in glaucoma . Am J Ophthalmol 63:982-989, 1967. 2. Oosterhuis JA, Gortzak-Moorstein N: Fluorescein angiography of the optic disc in glaucoma . Ophthalmologica 160:331-353, 1970.Crossref 3. Begg IS, Drance SM, Goldman H: Fluorescein angiography in the evaluation of focal circulatory ischemia of the optic nervehead in relation to the arcuate scotoma in glaucoma . Can J Ophthalmol 7:68-74, 1972. 4. Spaeth G: Fluorescein angiography: Its contribution towards understanding the mechanisms of visual loss in glaucoma . Trans Am Ophthalmol Soc 73:491-553, 1975. 5. Schwartz B, Rieser JC, Fishbein SL: Fluorescein angiographic defects of the optic disc in glaucoma . Arch Ophthalmol 95:1961-1974, 1977.Crossref 6. Tsukahara S, Nagataki S, Sugaya M, et al: Visual field defects, cup-disc ratio and fluorescein angiography in glaucomatous optic atrophy . Adv Ophthalmol 35:73-93, 1978. 7. Loebl M, Schwartz B: Fluorescein angiographic defects of the optic disc in ocular hypertension . Arch Ophthalmol 95:1980-1984, 1977.Crossref 8. Evans PY, Shimizu K, Limaye S, et al: Fluorescein cineangiography of the optic nerve head . Trans Am Acad Ophthalmol Otolaryngol 77:OP260-273, 1973. 9. Fishbein SL, Schwartz B: Optic disc in glaucoma: Topography and extent of fluorescein filling defects . Arch Ophthalmol 95:1975-1979, 1977.Crossref 10. Allen L: Ocular fundus photography . Am J Ophthalmol 57:13-28, 1964. 11. Donaldson DD: A new camera for stereoscopic fundus photography . Trans Am Acad Ophthalmol 62:429-458, 1964. 12. Siegel S: Non-parametric Statistics for the Behavioral Sciences . New York, McGraw-Hill Book Co, 1956. 13. Hitchings RA, Spaeth GL: Fluorescein angiography in glaucoma , in Cant CJ (ed): Vision and Circulation . London, Henry Kimpton, 1976, pp 201-216. 14. Hitchings RA, Spaeth GL: Fluorescein angiography in chronic simple and low-tension glaucoma . Br J Ophthalmol 61:126-132, 1977.Crossref 15. Henkind P, Levitzky M: Angioarchitecture of the optic nerve: I. The papilla . Am J Ophthalmol 68:979-986, 1969. 16. Henkind P, Levitzky M: Angioarchitecture of the optic nerve: II. Lamina cribrosa . Am J Ophthalmol 68:986-995, 1969. 17. Anderson DR, Braverman S: Reevaluation of the optic disk vasculature . Am J Ophthalmol 82:165-174, 1976. 18. Lieberman MF, Maumenee AE, Green WR: Histologic studies of the vasculature of the anterior optic nerve . Am J Ophthalmol 82:405-423, 1976. 19. Hayreh SS: Structure and blood supply of the optic nerve , in Heilmann K, Richardson KT (eds): Glaucoma: Conceptions of a Disease . Philadelphia, WB Saunders Co, 1978, pp 78-96. 20. Villaseca A: The impact of intraocular pressure on the glaucomatous disk . Arch Ophthalmol 67:769-772, 1962.Crossref 21. Potts AM: Some rationalizations on chronic open-angle glaucoma . Am J Ophthalmol 86:743-755, 1978. 22. Emery JM, Landis D, Paton D, et al: The lamina cribrosa in normal and glaucomatous human eyes . Trans Am Acad Ophthalmol Otolaryngol 78:OP290-297, 1974.

Chandra, Suresh R.;Bresnick, George H.;Davis, Matthew D.;Miller, Steven A.;Myers, Frank

1980 Archives of Ophthalmology

doi: 10.1001/archopht.1980.01020040445010pmid: 6158936

Abstract • In eight patients with proliferative diabetic retinopathy treated with focal photocoagulation for preretinal new vessels, recurrent preretinal new-vessel systems supplied from the choroid developed. Subsequent treatment in most cases failed to produce permanent obliteration of the new-vessels networks. References 1. Little HL, Zweng HC: Complications of argon laser retinal photocoagulation . Trans Pac Coast Oto-ophthalmol Soc 52:115-129, 1971. 2. Patz A, Maumenee AE, Ryan SJ: Argon laser photocoagulation: Advantages and limitations . Trans Am Acad Ophthalmol Otolaryngol 75:569-579, 1971. 3. Zweng HC, Little HL, Hammond AH: Complications of argon laser photocoagulation . Trans Am Acad Ophthalmol Otolaryngol 78:195-204, 1974. 4. Galinos SO, Asdourian GK, Woolf MB, et al: Choroido-vitreal neovascularization after argon laser photocoagulation . Arch Ophthalmol 93:524-530, 1975.Crossref 5. Galinos SO, McMeel JW, Trempe CL, et al: Chorioretinal anastomoses after argon laser photocoagulation . Am J Ophthalmol 82:241-245, 1976. 6. Benson WE, Townsend RE, Pheasant TR: Choriovitreal and subretinal proliferations: Complications of photocoagulation . Ophthalmology 86:283-289, 1979.Crossref 7. Goldberg MF: Bruch's membrane and vascular growth . Invest Ophthalmol 15:443-446, 1976. 8. Wolf ED, Goldberg MF: Chorioretinal anastomoses resulting from photocoagulation in monkeys. Read before the Association for Research in Vision and Ophthalmology meeting in Sarasota, Fla, April-May, 1978. 9. Marshall J, Mellerio J: Histology of the formation of retinal laser lesions . Exp Eye Res 6:4-9, 1967.Crossref