Cancer Causes & Control

Liu, Tao; Xu, Qin-er; Zhang, Chuan-hui; Zhang, Peng

doi: 10.1007/s10552-013-0283-0pmid: 24026192

We found an excess risk of multiple myeloma. The non-Hodgkin’s lymphoma and leukemia that have shown weak effects should be investigated further.

Anic, Gabriella; Madden, Melissa; Thompson, Reid; Nabors, L.; Olson, Jeffrey; LaRocca, Renato; Browning, James; Brockman, John; Forsyth, Peter; Egan, Kathleen

doi: 10.1007/s10552-013-0281-2pmid: 23996192

The results do not support a role for body iron stores as a determinant of glioma risk.

Makar, Karen; Poole, Elizabeth; Resler, Alexa; Seufert, Brenna; Curtin, Karen; Kleinstein, Sarah; Duggan, David; Kulmacz, Richard; Hsu, Li; Whitton, John; Carlson, Christopher; Rimorin, Christine; Caan, Bette; Baron, John; Potter, John;

Prizment, Anna; Folsom, Aaron; Dreyfus, Jill; Anderson, Kristin; Visvanathan, Kala; Joshu, Corinne; Platz, Elizabeth; Pankow, James

doi: 10.1007/s10552-013-0285-ypmid: 24036889

The study corroborates a causative role of chronic low-grade inflammation in colorectal carcinogenesis.

Tsui, Jennifer; Rodriguez, Hector; Gee, Gilbert; Escobedo, Loraine; Kominski, Gerald; Bastani, Roshan

doi: 10.1007/s10552-013-0286-xpmid: 24043448

The majority of high-risk neighborhoods were geographically near safety-net clinics with HPV vaccination services. Due to low rates of vaccination, these findings suggest that while services are geographically accessible, additional efforts are needed to improve uptake. Programs aimed to increase awareness about the vaccine and to link underserved groups to vaccination services are warranted.

Valery, Patricia; Youlden, Danny; Baade, Peter; Ward, Leisa; Green, Adele; Aitken, Joanne

doi: 10.1007/s10552-013-0287-9pmid: 24036890

Differences in place of residence, socio-economic disadvantage, and cancer diagnostic group only partially explain the survival disadvantage of Indigenous children. Other reasons underlying the disparities in childhood cancer outcomes by Indigenous status are yet to be determined, but may involve factors such as differences in treatment.

Perrigue, Martine; Kantor, Elizabeth; Hastert, Theresa; Patterson, Ruth; Potter, John; Neuhouser, Marian; White, Emily

doi: 10.1007/s10552-013-0288-8pmid: 24057417

The weak inverse association observed between eating frequency and CRC is consistent with findings from other prospective studies. Modification of this relationship by diet quality and participant characteristics should be considered in the future studies.

Suzuki, Reiko; Iwasaki, Motoki; Hara, Azusa; Inoue, Manami; Sasazuki, Shizuka; Sawada, Norie; Yamaji, Taiki; Shimazu, Taichi; Tsugane, Shoichiro

doi: 10.1007/s10552-013-0289-7pmid: 24091793

Our results suggest an overall null association between total fruit and vegetable intake and breast cancer risk. Intake of cruciferous vegetable showed a statistically significant association with a decreased risk of breast cancer among premenopausal women.

Gialamas, Spyros; Sergentanis, Theodoros; Antonopoulos, Constantine; Dessypris, Nick; Chrousos, George; Petridou, Eleni

doi: 10.1007/s10552-013-0290-1pmid: 24085585

Meta-analysis of current evidence suggests positive association of serum leptin with CRA but not with CRC risk. Given the case–control nature of available studies, the limited number of studies on serum leptin and CRA, and the heterogeneity of CRC studies, carefully designed, prospective studies preferably reporting RRs adjusted for a variety of confounders may be warranted.

Möller, Elisabeth; Adami, Hans-Olov; Mucci, Lorelei; Lundholm, Cecilia; Bellocco, Rino; Johansson, Jan-Erik; Grönberg, Henrik; Bälter, Katarina

doi: 10.1007/s10552-013-0291-0pmid: 24048969

Our comprehensive life-course approach revealed no convincing associations between anthropometric measures and prostate cancer risk. However, we found some leads that deserve further investigation, particularly for early-life body size. Our study highlights the importance of the time window of exposure in prostate cancer development.