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for: activated abl kinase inhibits oncogenic transforming growth factor
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Activated Abl kinase inhibits oncogenic transforming growth factor-β signaling and tumorigenesis in mammary tumors
Allington, Tressa M.; Galliher-Beckley, Amy J.; Schiemann, William P.
Follow The FASEB Journal , Volume 23 (12): 4231
Fed of American Socs for Experimental Biology – Dec 1, 2009mammary tumors.—Allington, T. M., Galliher-Beckley, A. J., Schiemann, W. P. Activated Abl kinase inhibits oncogenic transforming growth factor-β signaling and tumorigenesis in mammary tumors. Abstract ...
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Fumarase tumor suppressor gene and MET oncogene cooperate in upholding transformation and tumorigenesis
Costa, Barbara; Dettori, Daniela; Lorenzato, Annalisa; Bardella, Chiara; Coltella, Nadia; Martino, Cosimo; Cammarata, Cristina; Carmeliet, Peter; Olivero, Martina; Di Renzo, Maria Flavia
Follow The FASEB Journal , Volume 24 (8): 2680
Fed of American Socs for Experimental Biology – Aug 1, 2010fibroblasts (MEFs) to model transformation, we found that fh knockdown results in increased expression of the met oncogene-encoded tyrosine kinase receptor through hypoxia-inducible factor (hif) stabilization ...
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AHI-1 interacts with BCR-ABL and modulates BCR-ABL transforming activity and imatinib response of CML stem/progenitor cells
Zhou, Liang L.; Zhao, Yun; Ringrose, Ashley; DeGeer, Donna; Kennah, Erin; Lin, Ann E.-J.; Sheng, Guoqing; Li, Xiao-Jiang; Turhan, Ali; Jiang, Xiaoyan
Follow The Journal of Experimental Medicine , Volume 205 (11): 2657
Rockefeller University Press – Oct 27, 2008, coexpression of Ahi-1 in BCR-ABL –inducible cells reverses growth deficiencies exhibited by BCR-ABL down-regulation and is associated with sustained phosphorylation of BCR-ABL and enhanced activation of JAK2 ...
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Oncogenic protein tyrosine kinases
G., Saglio; D., Cilloni
Follow Cellular and Molecular Life Sciences , Volume 61 (23)
Springer Journals – Dec 1, 2004activity. Although we still lack a complete definition of all the transformation pathways activated by Bcr-Abl, the recent introduction into clinical practice of tyrosine kinase inhibitor represents a major ...
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Oncogenic protein tyrosine kinases
M., Santoro; F., Carlomagno; R., Melillo; A., Fusco
Follow Cellular and Molecular Life Sciences , Volume 61 (23)
Springer Journals – Dec 1, 2004]. Intriguingly, RET can be activated independently of the GFL-GFR-a complexes. Nerve growth factor (NGF)-triggered activation of the TrkA kinase receptor promotes RET phosphorylation through GFL-independent ...
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Inhibition and Termination of Physiological Responses by GTPase Activating Proteins
Ligeti, Erzsébet; Welti, Stefan; Scheffzek, Klaus
Follow Physiological Reviews , Volume 92 (1): 237
The American Physiological Society – Jan 1, 2012migration, whereas inhibition of Cdc42 activation transforms directed migration into random movements ( 11 ). Finally, retraction of the tail involves contraction by the actomyosin complex, where a kinase ...
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Oncogenic protein tyrosine kinases
S., Ménard; P., Casalini; M., Campiglio; S., Pupa; E., Tagliabue
Follow Cellular and Molecular Life Sciences , Volume 61 (23)
Springer Journals – Dec 1, 2004to the HER family of receptor tyrosine kinases. In normal cells, activation of this receptor tyrosine kinase family triggers a rich network of signaling pathways that control normal cell growth ...
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A small molecule blocking oncogenic protein EWS-FLI1 interaction with RNA helicase A inhibits growth of Ewing's sarcoma
Erkizan, Hayriye V; Kong, Yali; Merchant, Melinda; Schlottmann, Silke; Barber-Rotenberg, Julie S; Yuan, Linshan; Abaan, Ogan D; Chou, Tsu-hang; Dakshanamurthy, Sivanesan; Brown, Milton L; Üren, Aykut; Toretsky, Jeffrey A
Follow Nature Medicine , Volume 15 (7)
Nature Publishing Group (NPG) – Jul 5, 2009, such as breakpoint cluster region–Abelson oncoprotein (BCR-ABL) , lead to constitutively activated kinases, the majority lead to fusion proteins that function as transcription factors and lack intrinsic enzymatic ...
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Impact of interactions between normal and transformed epithelial cells and the relevance to cancer
Catherine, Hogan
Follow Cellular and Molecular Life Sciences , Volume 69 (2)
Springer Journals – Jan 1, 2012gap junction function and activity, normal cells were no longer able to inhibit transformed cell growth [12, 13]. Gap junctions are deï¬ned as cellâcell junctions between two apposing plasma ...
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Functional differences between two classes of oncogenic mutation in the PIK3CA gene
Chaussade, Claire; Cho, Kitty; Mawson, Claire; Rewcastle, Gordon W.; Shepherd, Peter R.
Follow Biochemical and Biophysical Research Communications , Volume 381 (4)
Elsevier – Apr 17, 2009helical domain mutants are in a state mimicking activation by growth factors whereas kinase domain mutants mimic the state activated by H-Ras. PI 3-kinases phosphorylate the D-3 position on inositol ...
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