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Downloaded from ajplung.physiology.org January 21, 2011 AJP - Lung Cellular and Molecular Physiology publishes original research covering the broad scope of molecular, cellular, and integrative aspects of normal and abnormal functi of cells and compents of the respiratory system. It is published 12 times a year (mthly) by the American Physiological Society, 9650 Rockville Pike, Bethesda MD 20814-3991. Copyright © 1997 by the American Physiological Society. ISSN: 1040-0605, ESSN: 1522-1504. Visit our website at http://www.the-aps.org/. AT-acetylcysteine after prolged does not protect against type II cell injury exposure to hyperoxia in rats ROB J. VAN KLAVEREN, DAVID DINSDALE, JAN L. PYPE, MAURITS DEMEDTS, AND BENOIT NEMERY Laboratory of Pneumology, Unit of Lung Toxicology and Unit of Pharmacology, Katholieke Universiteit Leuven, B-3000 Leuven, Belgium; and Medical Research Council Toxicology Unit, Leicester LEl 9HN, United Kingdom van Klaveren, Rob J., David Dinsdale, Jan L. Pype, Maurits Demedts, and Benoit Nemery. N-acetylcysteine does not protect against type II cell injury after prolged exposure to hyperoxia in rats. Am. J. Physiol. 273 (Lung CeZZ. MOL. PhysioZ. 17): L548-L555, 1997. -Although the antioxidant properties of N-acetylcysteine (NAC) in vitro are widely accepted, the efficacy in the preventi of 02 in vivo is poorly documented.
AJP - Lung Cellular and Molecular Physiology – The American Physiological Society
Published: Sep 1, 1997
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