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E. Sowton, D. Gupta, I. Baker (1975)
Comparative effects of beta adrenergic blocking drugs.Thorax, 30
Hans Astrom, Hans Vallin (1974)
Effect of a new beta-adrenergic blocking agent, ICI 66o82, on exercise haemodynamics and airway resistance in angina pectoris.British Heart Journal, 36
C. Kumana, G. Marlin, C. Kaye, D. Smith (1974)
New Approach to Assessment of Cardioselectivity of Beta-blocking DrugsBritish Medical Journal, 4
P. Roy, L. Day, E. Sowton (1975)
Effect of new beta-adrenergic blocking agent, Atenolol (Tenormin), on pain frequency, trinitrin consumption, and exercise ability.British Medical Journal, 3
A. Barrett, J. Carter, J. Fitzgerald, R. Hull, D. Count* (1973)
A new type of cardioselective adrenoceptive blocking drug.British journal of pharmacology, 48 2
H. Åström (1975)
Comparison of the effects on airway conductance of a new selective beta-adrenergic blocking drug, atenolol, and propranolol in asthmatic subjects.Scandinavian journal of respiratory diseases, 56 6
J. Petrie, D. Galloway, J. Webster, W. Simpson, J. Lewis (1975)
Atenolol and bendrofluazide in hypertension.British Medical Journal, 4
(1972)
Significance of beta blockade on ventilatory function in normal and asthmatic subjects
R. Jones (1972)
Significance of effect of beta blockade on ventilatory function in normal and asthmatic subjectsThorax, 27
L. Hansson, H. Aberg, B. Karlberg, A. Westerlund (1975)
Controlled study of atenolol in treatment of hypertension.British Medical Journal, 2
G. Addis, J. Thorp (1976)
Effects of oxprenolol on the airways of normal and bronchitic subjectsEuropean Journal of Clinical Pharmacology, 9
Beumer Hm, H. Hardonk (1972)
Effects of beta-adrenergic blocking drugs on ventilatory function in asthmaticsEuropean Journal of Clinical Pharmacology, 5
228 11 11 1 1 N. P. Boye J. R. Vale University Department of Lung Diseases, Rikshospitalet Oslo Norway Summary The bronchial effect of intravenous atenolol (ICI 66.082) has been studied in a doubleblind cross over trial in 10 patients with pronounced, labile bronchial asthma. A single i. v. dose of atenolol 3 mg, sufficient to cause a fall in heart rate and systolic blood pressure at rest, induced only a slight and clinically almost negligible impairment of ventilatory function. An ordinary therapeutic dose of salbutamol by inhalation far outweighed the bronchial effect of atenolol. The drug appears promising with regard to its cardio-selective properties.
European Journal of Clinical Pharmacology – Springer Journals
Published: Jan 1, 1977
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