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- Publisher
- Oxford University Press
- Copyright
- Copyright © 2015 Oxford University Press
- ISSN
- 0143-3334
- eISSN
- 1460-2180
- DOI
- 10.1093/carcin/23.4.667
- Publisher site
- See Article on Publisher Site
Abstract
Association of CYP2C9 genotypes leading to high enzyme activity and colorectal cancer risk deepdyve.host = 'www.deepdyve.com'; // replace with an affiliateId that we provide deepdyve.affiliateId = "highwire-oupjournals"; // the div to fill in with the rental link deepdyve.divIdList = ('rentalLink', 'rentalLink2'); // this is the anchor text for the rental link deepdyve.rentText = "Rent Article at DeepDyve"; deepdyve.divIdMap = {'rentalLink':'Rent Article at DeepDyve','rentalLink2':'Learn more here.'}; // document identifier and identifier type deepdyve.fieldName = 'journal_doi'; deepdyve.docId = "10.1093/carcin/23.4.667"; var callbackToken='505C503DFC0FA97'; var subCode='oupjournal_sub'; var gAuthTimeStamp = '2015-10-29T01:12:43.162-07:00'; var gSessionId = 'xNF1An6YbHSxaygkYmn48Q'; var gAuthzRequired = 'false'; var gAuthnMethods1 = 'ip'; var gAuthnMethods2 = 'ip'; var gAuthnIPs = '64.13.143.102'; var gAuthnIndividuals = ''; var gAuthnInstitutions = '151647456'; MathJax.Hub.Config({ tex2jax: { inlineMath: (("$","$"),("\\(","\\)")), processClass: "tex2jax_process|mathjax" } }); MathJax.Hub.Queue(function() { gColTempResize = true; fixColHeights(1); gColTempResize = false; }); var siqDOI = encodeURIComponent("10.1093/carcin/23.4.667"); var siqIsOpenAccess = encodeURIComponent(""); var siqPubDate = encodeURIComponent("20020401"); if (siqDOI.length == 0) { siqDOI = "UNKNOWN"; } if (gAuthnIndividuals.length != 0) { if (gAuthnInstitutions.length != 0) { authnEntity = encodeURIComponent(gAuthnIndividuals + ',' + gAuthnInstitutions); } else { authnEntity = encodeURIComponent(gAuthnIndividuals); } } else { authnEntity = encodeURIComponent(gAuthnInstitutions); } var commonString = 'authSessionId=' + gSessionId + String.fromCharCode(0x26) + 'authzRequired=' + gAuthzRequired + String.fromCharCode(0x26) + 'authentication_method=' + encodeURIComponent(gAuthnMethods2) + String.fromCharCode(0x26) + 'authnIPs=' + gAuthnIPs + String.fromCharCode(0x26) + 'authnInstitutions=' + authnEntity; var gPageId = "pageid-content"; var gVariant = "extract"; // Not completely done var eventType = "extract"; var accessType; if (siqIsOpenAccess == 'true') { accessType = 'SOA'; } else { accessType = 'subscription'; } var NTPT_PGEXTRA = commonString + String.fromCharCode(0x26) + 'event_type=' + eventType + String.fromCharCode(0x26) + 'publication_date=' + siqPubDate + String.fromCharCode(0x26) + 'access_type=' + accessType + String.fromCharCode(0x26) + 'doi=' + siqDOI ; // alert("NTPT_PGEXTRA is " + NTPT_PGEXTRA); We use cookies to enhance your experience on our website. By continuing to use our website, you are agreeing to our use of cookies. You can change your cookie settings at any time. Find out more Skip Navigation Oxford Journals Contact Us My Basket My Account Carcinogenesis About This Journal Contact This Journal Subscriptions View Current Issue (Volume 36 Issue 10 October 2015) Archive Search Institution: :: Sign In as Personal Subscriber Oxford Journals Medicine & Health Science & Mathematics Carcinogenesis Volume 23 Issue 4 Pp. 667-668. Association of CYP2C9 genotypes leading to high enzyme activity and colorectal cancer risk Carmen Martínez 1 , Elena García-Martín 2 , José M. Ladero 3 , Javier Sastre 4 , Francisco Garcia-Gamito 5 , Manuel Diaz-Rubio 3 and José A.G. Agúndez 1, , 6 1 Department of Pharmacology, Medical School and 2 Department of Biochemistry, School of Biological Sciences, University of Extremadura, Avda. de Elvas s/n. E-06071, Badajoz, Spain, 3 Service of Gastroenterology and 4 Medical Oncology, San Carlos University Hospital, Prof. Martin Lagos s/n, E-28040, Madrid, Spain and 5 Service of Surgery, Hospital INSALUD, Pol Nueva Ciudad s/n E-06800 Mérida, Spain Received January 31, 2002. Accepted January 31, 2002. Dear Sir, Thank you for the opportunity to reply the letter from Yasar et al . In their letter, Yasar et al ., expressed concerns regarding the selection of the control group in our previous study ( 1 ). With regard to the first matter addressed i.e., that patients with benign diseases of the colon are more adequate controls than healthy subjects we do not agree for the following reasons. From a clinical point of view it has little sense to compare cancer patients with patients with benign diseases. According to the suggestion from Yasar et al ., a control group could have been composed from individuals with benign polyps, ulcerative colitis and irritable bowel syndrome among the most prevalent benign diseases of the colon. However, colorectal polyps may be pre-cancerous and the risk to develop colorectal cancer is much higher in subjects with polyps than in the general population. In fact it has been reported that ~95% of colorectal cancers arise in benign adenomatous polyps ( 2 ). This makes this subgroup inadequate for comparison. Ulcerative colitis … (Full Text of this Article) « Previous | Next Article » Table of Contents This Article Carcinogenesis (2002) 23 (4): 667-668. doi: 10.1093/carcin/23.4.667 » Extract Free Full Text (HTML) Free Full Text (PDF) Free Classifications RESPONSE TO LETTER TO THE EDITOR Services Article metrics Alert me when cited Alert me if corrected Find similar articles Similar articles in Web of Science Add to my archive Download citation Request Permissions Citing Articles Load citing article information Citing articles via CrossRef Citing articles via Scopus Citing articles via Web of Science Citing articles via Google Scholar Google Scholar Articles by Martínez, C. Articles by Agúndez, J. A. Search for related content PubMed Articles by Martínez, C. Articles by García-Martín, E. Articles by Ladero, J. M. Articles by Sastre, J. Articles by Garcia-Gamito, F. Articles by Diaz-Rubio, M. Articles by Agúndez, J. A. 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Journal
Carcinogenesis – Oxford University Press
Published: Apr 1, 2002