Prevention and reversal of renal injury by leptin in a new mouse model of diabetic nephropathy Takayoshi Suganami * , 1 , Masashi Mukoyama * ,2 , Kiyoshi Mori * , Hideki Yokoi * , Masao Koshikawa * , Kazutomo Sawai * , Shuji Hidaka * , Ken Ebihara * , Tomohiro Tanaka * , Akira Sugawara * , Hiroshi Kawachi † , Charles Vinson ‡ , Yoshihiro Ogawa * , 1 and Kazuwa Nakao * * Department of Medicine and Clinical Science, Kyoto University Graduate School of Medicine, Kyoto, Japan; † Department of Cell Biology, Institute of Nephrology, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan; and ‡ Laboratory of Biochemistry, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, USA. 2 Correspondence: Department of Medicine and Clinical Science, Kyoto University Graduate School of Medicine, 54 Shogoin Kawahara-cho, Sakyo-ku, Kyoto 606-8507, Japan. E-mail: muko@kuhp.kyoto-u.ac.jp <h3>SPECIFIC AIMS</h3> There has been a long debate about the reversibility of diabetic nephropathy, and the problem has been difficult to approach because of the lack of suitable animal diabetic nephropathy models closely resembling human diabetic glomerulosclerosis. We investigated and evaluated whether renal injury in a mouse model of lipoatrophic
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Prevention and reversal of renal injury by leptin in a new mouse model of diabetic nephropathy
Suganami, Takayoshi; Mukoyama, Masashi; Mori, Kiyoshi; Yokoi, Hideki; Koshikawa, Masao; Sawai, Kazutomo; Hidaka, Shuji; Ebihara, Ken; Tanaka, Tomohiro; Sugawara, Akira; Kawachi, Hiroshi; Vinson, Charles; Ogawa, Yoshihiro; Nakao, Kazuwa
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, Volume 19 (1): 127
Fed of American Socs for Experimental Biology – Jan 1, 2005
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