Lipoprotein(a) in atherosclerotic plaques recruits inflammatory cells through interaction with Mac-1 integrin Sotirios N. Sotiriou † , Valeria V. Orlova * ,† , Nadia Al-Fakhri ‡ , Eveliina Ihanus § , Matina Economopoulou * , Berend Isermann † , Khalil Bdeir || , Peter P. Nawroth † , Klaus T. Preissner ¶ , Carl G. Gahmberg § , Marlys L. Koschinsky ** and Triantafyllos Chavakis * ,† ,1 * Experimental Immunology Branch, NCI, NIH, Bethesda, Maryland, USA; † Department of Internal Medicine I, University Heidelberg, Heidelberg, Germany; ‡ Department of Clinical Chemistry and Molecular Diagnostics, Philipps-University, Marburg, Germany; § Division of Biochemistry, Faculty of Biosciences, University of Helsinki, Finland; || Department of Pathology and Laboratory Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, USA; ¶ Institute for Biochemistry, Justus-Liebig-University, Giessen, Germany; and ** Department of Biochemistry, Queen's University, Kingston, Ontario, Canada 1 Correspondence: Experimental Immunology Branch, NCI, NIH, 10 Center Drive, Rm. 4B17, Bethesda, MD 20892, USA. E-mail: chavakist@mail.nih.gov <h3>SPECIFIC AIMS</h3> Lipoprotein(a) [Lp(a)], consisting of LDL and apolipoprotein(a) [apo(a)], which contains multiple repeats resembling plasminogen kringle 4, is considered a risk factor for the development of atherosclerotic disorders, however, the underlying mechanisms for the atherogenicity of Lp(a) are not completely
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Lipoprotein(a) in atherosclerotic plaques recruits inflammatory cells through interaction with Mac-1 integrin
Sotiriou, Sotirios N.; Orlova, Valeria V.; Al-Fakhri, Nadia; Ihanus, Eveliina; Economopoulou, Matina; Isermann, Berend; Bdeir, Khalil; Nawroth, Peter P.; Preissner, Klaus T.; Gahmberg, Carl G.; Koschinsky, Marlys L.; Chavakis, Triantafyllos
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, Volume 20 (3): 559
Fed of American Socs for Experimental Biology – Mar 1, 2006
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