Hydrogen sulfide is a novel mediator of lipopolysaccharide-induced inflammation in the mouse Ling Li * , Madhav Bhatia * , Yi Zhun Zhu * , Yi Chun Zhu † , Raina Devi Ramnath * , Zhong Jing Wang * , Farhana Binte Mohammed Anuar * , Matthew Whiteman ‡ , Manuel Salto-Tellez § and Philip K. Moore * ,1 * Department of Pharmacology, National University of Singapore, Singapore; † Department of Physiology and Pathophysiology, Key Laboratory of Molecular Medicine of The Ministry of Education, Fudan University Shanghai Medical College, Shanghai, People’s Republic of China; ‡ Department of Biochemistry, National University of Singapore, Singapore; § Department of Pathology, National University Hospital, Singapore 1 Correspondence: Department of Pharmacology, National University of Singapore, Block MD2, 18 Medical Dr., Singapore, 117597. E-mail: phchead@nus.edu.sg <h3>SPECIFIC AIMS</h3> The aim of this work was to investigate the part played by the novel gaseous mediator, hydrogen sulfide (H 2 S) in E. coli lipopolysaccharide-induced inflammation in the mouse and to examine plasma concentrations of this mediator in patients with septic shock. <h3>PRINCIPAL FINDINGS</h3> 1. LPS-induced endotoxic shock is associated with increased plasma H 2 S concentration and tissue H 2 S tissue synthesizing activity LPS administration
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Hydrogen sulfide is a novel mediator of lipopolysaccharide-induced inflammation in the mouse
Li, Ling; Bhatia, Madhav; Zhu, Yi Zhun; Zhu, Yi Chun; Ramnath, Raina Devi; Wang, Zhong Jing; Anuar, Farhana Binte Mohammed; Whiteman, Matthew; Salto-Tellez, Manuel; Moore, Philip K.
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, Volume 19 (9): 1196
Fed of American Socs for Experimental Biology – Jul 1, 2005
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