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Journals The FASEB Journal Volume 19 Issue 12

Erythropoietin is neuroprotective, improves functional recovery, and reduces neuronal apoptosis and inflammation in a rodent model of experimental closed head injury I. Yatsiv * ,† , N. Grigoriadis ‡ , C. Simeonidou ‡ , P. F. Stahel § , O. I. Schmidt § , A. G. Alexandrovitch * , J. Tsenter * ,|| and E. Shohami * ,1 * Departments of Pharmacology, † Pediatric Intensive Care and || Rehabilitation, Hebrew University Hadassah Medical Center, Jerusalem, Israel; ‡ Department of Neurology AHEPA University Hospital and ‡Department of Physiology, Faculty of Medicine, Aristotle University Thessaloniki, Greece; and § Department of Trauma and Reconstructive Surgery, Charité University Medical School, Berlin, Germany 1 Correspondence: Department of Pharmacology, School of Pharmacy, The Hebrew University of Jerusalem, Jerusalem 91120, Israel. E-mail: esty@cc.huji.ac.il <h3>SPECIFIC AIMS</h3> To date there are no pharmacological agents to treat traumatic brain injury. The present study was aimed to demonstrate the protective effect of erythropoietin (EPO) treatment after brain injury in the mouse. We assessed the effect of treatment at the neurobehavioral and cognitive level, as well as at tissue level (reduced inflammation) and cellular level (less neuronal apoptosis). <h3>PRINCIPAL FINDINGS</h3> <h3>1. Effect of posttraumatic rhEpo treatment on the neurobehavioral recovery</h3>

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Erythropoietin is neuroprotective, improves functional recovery, and reduces neuronal apoptosis and inflammation in a rodent model of experimental closed head injury

Yatsiv, I.; Grigoriadis, N.; Simeonidou, C.; Stahel, P. F.; Schmidt, O. I.; Alexandrovitch, A. G.; Tsenter, J.; Shohami, E.
The FASEB Journal , Volume 19 (12): 1701
Fed of American Socs for Experimental Biology – Oct 1, 2005

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