Disuse atrophy and exercise rehabilitation in humans profoundly affects the expression of genes associated with the regulation of skeletal muscle mass 1 SIMON W. JONES * ,2 , ROGER J. HILL † , PHILIP A. KRASNEY † , BARBARA O’CONNER † , NICHOLAS PEIRCE * and PAUL L. GREENHAFF * Centre for Integrated Systems Biology and Medicine, School of Biomedical Sciences, * The Medical School, Queen’s Medical Centre, University of Nottingham, Nottingham, UK; and † Department of Cardiovascular & Metabolic Diseases, Pfizer Global Research and Development, Groton, Connecticut, USA 2 Correspondence: Centre for Integrated Systems Biology and Medicine, School of Biomedical Sciences, The Medical School, Queen’s Medical Centre, University of Nottingham, Nottingham, NG7 2UH, UK. E-mail: simonwynjones2003@yahoo.co.uk <h3>SPECIFIC AIMS</h3> After injury such as head trauma or stroke and during illness such as cancer and AIDS there is significant loss of skeletal muscle mass, paralleled by a loss of muscle function. The aim of this study was to provide a comprehensive profiling of a number of candidate genes associated with atrophy and anabolism during immobilization and subsequent exercise rehabilitation and relate these changes to measurements of muscle mass and function in humans, thereby gaining clearer insights into the pathways
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Disuse atrophy and exercise rehabilitation in humans profoundly affects the expression of genes associated with the regulation of skeletal muscle mass
JONES, SIMON W.; HILL, ROGER J.; KRASNEY, PHILIP A.; OCONNER, BARBARA; PEIRCE, NICHOLAS; GREENHAFF, PAUL L.
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, Volume 18 (9): 1025
Fed of American Socs for Experimental Biology – Jun 1, 2004
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