Deficiency of polycystin-2 reduces Ca 2+ channel activity and cell proliferation in ADPKD lymphoblastoid cells 1 GIANLUCA AGUIARI, MANUELA BANZI, STEFANIA GESSI * , YIQIANG CAI ‡ , EMANUELA ZEGGIO † , ELISA MANZATI, ROBERTA PIVA, ELISABETTA LAMBERTINI † , LUISA FERRARI || , DORIEN J. PETERS § , FRANCESCO LANZA || , PETER C. HARRIS ** , PIER ANDREA BOREA * , STEFAN SOMLO ‡ and LAURA DEL SENNO 2 Department of Biochemistry and Molecular Biology, Departments of * Clinical and Experimental Medicine, Pharmacology Unit, || Biomedical Sciences and Advanced Therapies, Haematology Unit, and † Biotechnology Center, University of Ferrara, Ferrara, Italy; § Department of Human and Clinical Genetics, Leiden University Medical Center, Leiden, The Netherlands; ** Department of Pathology, Mayo Clinic, Rochester, Minnesota, USA; and ‡ Departments of Internal Medicine and Genetics, Yale University School of Medicine, New Haven, Connecticut, USA 2 Correspondence: Department of Biochemistry and Molecular Biology, Via L. Borsari 46, 44100 Ferrara, Italy. E-mail: sen@unife.it <h3>SPECIFIC AIMS</h3> Polycystin-2 (PC2), the product of the PKD2 gene mutated in type 2 autosomal-dominant polycystic kidney disease (ADPKD), is a Ca 2+ -permeable cation channel found in kidney epithelia and other tissues. The precise expression and subcellular
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Deficiency of polycystin-2 reduces Ca2+ channel activity and cell proliferation in ADPKD lymphoblastoid cells
AGUIARI, GIANLUCA; BANZI, MANUELA; GESSI, STEFANIA; CAI, YIQIANG; ZEGGIO, EMANUELA; MANZATI, ELISA; PIVA, ROBERTA; LAMBERTINI, ELISABETTA; FERRARI, LUISA; PETERS, DORIEN J.; LANZA, FRANCESCO; HARRIS, PETER C.; BOREA, PIER ANDREA; SOMLO, STEFAN; DEL SENNO, LAURA
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, Volume 18 (7): 884
Fed of American Socs for Experimental Biology – May 1, 2004
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