Age-associated increase in oxidative stress and nuclear factor κB activation are attenuated in rat liver by regular exercise 1 ZSOLT RADÁK * ,2 , HAE YOUNG CHUNG † ,2 , HISASHI NAITO ‡ , RYOYA TAKAHASHI § , KYUNG JIN JUNG † , HYON-JEEN KIM † and SATARO GOTO § * Laboratory of Exercise Physiology, School of Sport Science, Semmelweis University, Budapest, Hungary; † Department of Pharmacy, Pusan National University, Pusan, Korea; ‡ Department of Sports Physiology, School of Sports Sciences, Juntendo University, Tokyo, Japan; and § Departiment of Biochemistry, Faculty of Pharmaceutical Sciences, Toho University, Funabashi, Japan 2 Correspondence to: E-mail: radak@mail.hupe.hu or hyjung@pusan.ac.kr <h3>SPECIFIC AIMS</h3> The oxidative stress theory argues that senescent phenotypes (i.e.the intrinsic physiological and biochemical decline with age resulting in decreased survival) are primarily due to the accumulation of oxidative damage to cellular components. Environmental interactions that increase the ability of an organism to either decrease the production of ROS or increase the activity and effectiveness of antioxidant systems, have the potential to retard aging and increase average and/or maximal life span. One such environmental intervention that remains equivocal with respect to its effects on ROS production is regular physical exercise. Nuclear factor
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Age-associated increase in oxidative stress and nuclear factor κB activation are attenuated in rat liver by regular exercise
RADÁK, ZSOLT; CHUNG, HAE YOUNG; NAITO, HISASHI; TAKAHASHI, RYOYA; JUNG, KYUNG JIN; KIM, HYON-JEEN; GOTO, SATARO
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, Volume 18 (6): 749
Fed of American Socs for Experimental Biology – Apr 1, 2004
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