A role for endocannabinoids in the generation of parkinsonism and levodopa-induced dyskinesia in MPTP-lesioned non-human primate models of Parkinson’s disease Mario van der Stelt * , 1 , 2 , Susan H. Fox † , 1 , Michael Hill ‡ , 3 , Alan R. Crossman ‡ , Stefania Petrosino * , Vincenzo Di Marzo * ,4 and Jonathan M. Brotchie § ,4 * Endocannabinoid Research Group, Istituto di Chimica Biomolecolare, Consiglio Nazionale delle Ricerche, Pozzuoli (NA), Italy; † Movement Disorders Clinic, Toronto Western Hospital, Toronto, Canada; ‡ Division of Neuroscience, School of Biological Sciences, University of Manchester, Manchester UK; and § Toronto Western Research Institute, Toronto Western Hospital, Toronto, Canada 4 Correspondence: J.M.B., Toronto Western Research Institute, MC 11-419, Toronto Western Hospital, 399, Bathurst St, Toronto ON, Canada M5T 2S8. E-mail: Brotchie@uhnres.utoronto.ca ; V.D.M., Istituto di Chimica Biomolecolare, Consiglio Nazionale delle Ricerche, Via Campi Flegrei 34, 80078, Pozzuoli (NA) Italy. E-mail: vdimarzo@icmib.na.cnr.it <h3>SPECIFIC AIMS</h3> The aim of the present study was to assess systematically in nonhuman primates the role of endocannabinoid stimulation of cannabinoid CB1 receptors in the generation of symptoms of parkinsonism and L-DOPA-induced dyskinesia (LID), the major complication of treatments for PD and the major
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A role for endocannabinoids in the generation of parkinsonism and levodopa-induced dyskinesia in MPTP-lesioned non-human primate models of Parkinsons disease
van der Stelt, Mario; Fox, Susan H.; Hill, Michael; Crossman, Alan R.; Petrosino, Stefania; Di Marzo, Vincenzo; Brotchie, Jonathan M.
Follow The FASEB Journal
, Volume 19 (9): 1140
Fed of American Socs for Experimental Biology – Jul 1, 2005
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